Ulrike B. Depner

Friedrich-Alexander-Universität Erlangen-Nürnberg — Germany

Medicine · Health Sciences

4h-index899citations4works0.02yr avg

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Research Topics

Pain Mechanisms and Treatments(3), Neuropeptides and Animal Physiology(3), Inflammatory mediators and NSAID effects(2), Anesthesia and Pain Management(1), Receptor Mechanisms and Signaling(1)

Publications4 total

Spinal inflammatory hyperalgesia is mediated by prostaglandin E receptors of the EP2 subtype

199 cited

Journal of Clinical Investigation·2005· 199 citedOpen Access

Spinal inflammatory hyperalgesia is mediated by prostaglandin E receptors of the EP2 subtype

34 cited

Journal of Clinical Investigation·2005· 34 citedOpen Access

GlyR α3: An Essential Target for Spinal PGE <sub>2</sub> -Mediated Inflammatory Pain Sensitization

601 cited

Science·2004· 601 cited

Normal sensitivity to acute pain, but increased inflammatory hyperalgesia in mice lacking the nociceptin precursor polypeptide or the nociceptin receptor

65 cited

European Journal of Neuroscience·2003· 65 cited

Frequent Co-authors

Hanns Ulrich Zeilhofer(4), Seifollah Ahmadi(3), Cornelia Heindl(3), H.-M. Reinold(3), Ulrike Müller(3), Kay Brune(3), Beate Layh(2), May Hamza(2), Andreas Pahl(2), Shuh Narumiya(2), Victoria L. Harvey(1), Heinz Wässle(1), Trevor G. Smart(1), Kirsten Harvey(1), Burkhard Schütz(1), Osama M. Abo‐Salem(1), Andreas Zimmer(1), Pierrick Poisbeau(1), Hans Welzl(1), David P Wolfer(1)