Brian K. England

University of Montana Western — United States

Biochemistry, Genetics and Molecular Biology · Life Sciences

17h-index3.2kcitations30works0.02yr avg
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Research Topics

Muscle metabolism and nutrition(13), Metabolism and Genetic Disorders(8), Adipose Tissue and Metabolism(6), Diet and metabolism studies(6), Mitochondrial Function and Pathology(5)

Publications30 total

Timetabling Courses in Single-Subject Intensive Block Scheduling: Challenges and Solutions
Journal of Block and Intensive Learning and Teaching·2024Open Access
Mutations in the <i>SPTLC1</i> gene are a cause of juvenile amyotrophic lateral sclerosis that may be amenable to serine supplementation
2019· 2 citedOpen Access
Toxins versus Abnormal Cell Metabolism in the Pathophysiology of Chronic Renal Failure1
Contributions to nephrology·2015· 2 cited
Mechanisms contributing to muscle-wasting in acute uremia
Journal of the American Society of Nephrology·1998· 53 cited
Predictors of the progression of renal disease in the Modification of Diet in Renal Disease Study
Kidney International·1997· 695 citedOpen Access
Signals regulating accelerated muscle protein catabolism in uremia.
PubMed·1997· 9 cited
Muscle wasting in insulinopenic rats results from activation of the ATP-dependent, ubiquitin-proteasome proteolytic pathway by a mechanism including gene transcription.
Journal of Clinical Investigation·1996· 346 citedOpen Access
Effects of dietary protein restriction on the progression of advanced renal disease in the modification of diet in renal disease study
American Journal of Kidney Diseases·1996· 338 cited
The acidosis of chronic renal failure activates muscle proteolysis in rats by augmenting transcription of genes encoding proteins of the ATP-dependent ubiquitin-proteasome pathway.
Journal of Clinical Investigation·1996· 451 citedOpen Access
Protein degradation and increased mRNAs encoding proteins of the ubiquitin-proteasome proteolytic pathway in BC3H1 myocytes require an interaction between glucocorticoids and acidification.
Proceedings of the National Academy of Sciences·1996· 92 citedOpen Access
Glucocorticoids and acidosis stimulate protein and amino acid catabolism in vivo
Kidney International·1996· 108 citedOpen Access
Experimental acidemia and muscle cell pH in chronic acidosis and renal failure
American Journal of Physiology-Cell Physiology·1995· 40 cited
Mechanisms causing muscle loss in chronic renal failure
American Journal of Kidney Diseases·1995· 31 cited
Rat muscle branched-chain ketoacid dehydrogenase activity and mRNAs increase with extracellular acidemia
American Journal of Physiology-Cell Physiology·1995· 68 cited
Acidosis and Glucocorticoids Interact to Provoke Muscle Protein and Amino Acid Catabolism
Blood Purification·1995· 15 cited
Title Page
Blood Purification·1995Open Access
Table of Contents
Blood Purification·1995Open Access
Author/Subject Indexes Vol. 13, No.3-4, 1995
Blood Purification·1995Open Access
Acidosis and glucocorticoids concomitantly increase ubiquitin and proteasome subunit mRNAs in rat muscle
American Journal of Physiology-Cell Physiology·1994· 184 cited
Metabolic acidosis stimulates muscle protein degradation by activating the adenosine triphosphate-dependent pathway involving ubiquitin and proteasomes.
Journal of Clinical Investigation·1994· 403 citedOpen Access
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Frequent Co-authors

William E. Mitch(18), S. Russ Price(15), James L. Bailey(8), Claudine Jurkovitz(5), R C May(4), R. G. Ebb(3), Barbara J. Ballermann(3), Mark J. Ott(3), Brian J.G. Pereira(3), Richard Bucala(3), Helen Vlassara(3), Beth Piraino(3), John M. Burkart(3), Vicki Rubin Kelley(3), Terry B. Strom(3), Lorna Paul(3), Paul W. Sanders(3), Thomas A. Golper(3), C. Patrick(3), L.V. McIntire(3)