Kun‐Yong Kim

Yale Cancer Center — United States

Medicine · Health Sciences

17h-index2.2kcitations43works0.02yr avg
OpenAlex↗ORCID↗Google Scholar↗Lab page↗

Accepting Students?

No reports yet. Know if this professor is taking students?

Research Topics

Adipokines, Inflammation, and Metabolic Diseases(14), Pluripotent Stem Cells Research(12), CRISPR and Genetic Engineering(9), Genetics and Neurodevelopmental Disorders(6), Autism Spectrum Disorder Research(4)

Publications43 total

Inflammation increases the penetrance of behavioral impairment in <i>Shank3</i> haploinsufficiency mice – can it explain the behavioral regression in Autism?
2025Open Access
Functional Validation of Alcohol Dependence-Associated <i>FYN</i> Variants Using Gene Editing and Stem Cell Study Approaches
International Journal of Stem Cells·2025Open Access
Female naïve human pluripotent stem cells carry X chromosomes with Xa-like and Xi-like folding conformations
Science Advances·2023· 7 citedOpen Access
Supplementary Figure Legends 1-4 from Adiponectin-Activated AMPK Stimulates Dephosphorylation of AKT through Protein Phosphatase 2A Activation
2023Open Access
Supplementary Figure 1 from Adiponectin-Activated AMPK Stimulates Dephosphorylation of AKT through Protein Phosphatase 2A Activation
2023Open Access
Supplementary Figure 2 from Adiponectin-Activated AMPK Stimulates Dephosphorylation of AKT through Protein Phosphatase 2A Activation
2023Open Access
Data from Adiponectin-Activated AMPK Stimulates Dephosphorylation of AKT through Protein Phosphatase 2A Activation
2023Open Access
Supplementary Figure 1 from Adiponectin-Activated AMPK Stimulates Dephosphorylation of AKT through Protein Phosphatase 2A Activation
2023Open Access
Supplementary Figure 2 from Adiponectin-Activated AMPK Stimulates Dephosphorylation of AKT through Protein Phosphatase 2A Activation
2023Open Access
Supplementary Figure Legends 1-4 from Adiponectin-Activated AMPK Stimulates Dephosphorylation of AKT through Protein Phosphatase 2A Activation
2023Open Access
Data from Adiponectin-Activated AMPK Stimulates Dephosphorylation of AKT through Protein Phosphatase 2A Activation
2023Open Access
Supplementary Figure 3 from Adiponectin-Activated AMPK Stimulates Dephosphorylation of AKT through Protein Phosphatase 2A Activation
2023Open Access
Supplementary Figure 4 from Adiponectin-Activated AMPK Stimulates Dephosphorylation of AKT through Protein Phosphatase 2A Activation
2023Open Access
Supplementary Table 1 from Adiponectin-Activated AMPK Stimulates Dephosphorylation of AKT through Protein Phosphatase 2A Activation
2023Open Access
Supplementary Table 1 from Adiponectin-Activated AMPK Stimulates Dephosphorylation of AKT through Protein Phosphatase 2A Activation
2023Open Access
Supplementary Figure 4 from Adiponectin-Activated AMPK Stimulates Dephosphorylation of AKT through Protein Phosphatase 2A Activation
2023Open Access
Supplementary Figure 3 from Adiponectin-Activated AMPK Stimulates Dephosphorylation of AKT through Protein Phosphatase 2A Activation
2023Open Access
Dysregulation of BRD4 Function Underlies the Functional Abnormalities of MeCP2 Mutant Neurons
Molecular Cell·2020· 85 citedOpen Access
Engineering of human brain organoids with a functional vascular-like system
Nature Methods·2019· 849 cited
The RNA exosome nuclease complex regulates human embryonic stem cell differentiation
The Journal of Cell Biology·2019· 50 citedOpen Access
page 1 of 3Next →

Frequent Co-authors

In‐Hyun Park(24), Jong‐Seok Lim(16), Young Yang(16), Keun Il Kim(15), Ahmi Baek(14), Jieun Hwang(14), Yeon A. Choi(14), Joon Jeong(14), Myeong‐Sok Lee(14), Dea Ho Cho(14), Yoshiaki Tanaka(13), Yangfei Xiang(9), Eriona Hysolli(9), Mei Zhong(6), Sherman M. Weissman(6), Benjamin Patterson(5), Bilal Çakir(4), Yong Wook Jung(4), Xinghua Pan(4), Young‐Jin Kang(3)