Nofar Rosenberg

Hadassah Medical Center — Israel

Biochemistry, Genetics and Molecular Biology · Life Sciences

8h-index421citations24works0.02yr avg
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Research Topics

MicroRNA in disease regulation(9), Liver Disease Diagnosis and Treatment(9), Liver physiology and pathology(6), Cancer-related molecular mechanisms research(5), Fibroblast Growth Factor Research(5)

Publications24 total

FRI-066 The tumor suppressor miR-122 regulates liver cancer stem cells immune evasion and proliferation through CD24
Journal of Hepatology·2025
FRI-064 MicroRNA-122 functions as a rheostat regulating liver tolerance, inflammation and fibrosis
Journal of Hepatology·2025
Abstract LB457: Suppressing liver metastasis growth by inducing the endogenous expression of the tumor suppressor miR-34a by small synthetic molecules
Cancer Research·2025
WED-534 MiR122 expression regulates liver immune tolerance and microenvironment
Journal of Hepatology·2024
Fibroblast growth factors induce hepatic tumorigenesis post radiofrequency ablation
Scientific Reports·2023· 8 citedOpen Access
Ductular reaction is mediated by CD24 and regulated by miR-122
Journal of Hepatology·2023
Supplementary Data from Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis
2023Open Access
Data from Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis
2023Open Access
Supplementary Data from Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis
2023Open Access
Data from Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis
2023Open Access
Supplementary Materials and Methods from Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis
2023Open Access
Supplementary Materials and Methods from Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis
2023Open Access
Combined hepatocellular-cholangiocarcinoma derives from liver progenitor cells and depends on senescence and IL-6 trans-signaling
Journal of Hepatology·2022· 62 citedOpen Access
MiR-122, the regulator of the immune privileged liver
Journal of Hepatology·2022
Mixed HCC-CCA originates from hepatic progenitor cells, is dependent on IL6 singling and is ablated by senolytic agents
Journal of Hepatology·2022
Multiple Roles of IL6 in Hepatic Injury, Steatosis, and Senescence Aggregate to Suppress Tumorigenesis
Cancer Research·2021· 30 citedOpen Access
The pro-oncogenic effect of the lncRNA H19 in the development of chronic inflammation-mediated hepatocellular carcinoma
Oncogene·2020· 33 citedOpen Access
Agonist of RORA Attenuates Nonalcoholic Fatty Liver Progression in Mice via Up-regulation of MicroRNA 122
Gastroenterology·2020· 85 citedOpen Access
THU-489-Mixed HCC-ICC liver cancer derives from hepatic progenitor cells: A lineage tracing investigation in a mouse liver inflammation model
Journal of Hepatology·2019Open Access
Mixed HCC-ICC liver cancer derives from hepatic progenitor cells- A lineage tracing investigation in mouse liver inflammation models
Journal of Hepatology·2018
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Frequent Co-authors

Eithan Galun(21), Amnon Peled(14), Stefan Rose‐John(14), Jonathan H. Axelrod(13), Hilla Giladi(12), Rifaat Safadi(12), Tali Lanton(11), Yuval Nevo(11), Rinat Abramovitch(11), Mathias Heikenwälder(9), Anat Shriki(9), Amir Sonnenblick(9), Daniel Goldenberg(8), Sharona Elgavish(8), Orit Pappo(8), Orr Levkovitch‐Siany(7), Dana Eidelshtein(7), Maytal Gefen(6), Matthias Van Haele(6), Tania Roskams(6)