Alberto Villanueva

Institut Català d'Ornitologia — Spain

Biochemistry, Genetics and Molecular Biology · Life Sciences

64h-index17.2kcitations495works1.72yr avg
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Research Topics

RNA modifications and cancer(81), Epigenetics and DNA Methylation(55), Cancer Genomics and Diagnostics(46), Cancer Research and Treatments(42), Genetic factors in colorectal cancer(38)

Publications495 total

Author response: Chromatin activity of IκBα mediates the exit from naïve pluripotency
2025Open Access
Specific targeting of the pseudogene RPSAP52 reduces ovarian tumor growth in patient-derived orthoxenograft models
European Journal of Cancer·2025· 1 cited
Chromatin activity of IκBα mediates the exit from naïve pluripotency
eLife·2025Open Access
Novel homozygous pathogenic variant in HSD11B2 as a cause of apparent mineralocorticoid excess
Journal of Hypertension·2025
Chromatin activity of IκBα mediates the exit from naïve pluripotency
2025Open Access
Cancer Exosomes Perform Cell-Independent MicroRNA Biogenesis and Promote Tumorigenesis
Cancer Cell·2025· 1 cited
A Genetic Defect in Exportin-5 Traps Precursor MicroRNAs in the Nucleus of Cancer Cells
Cancer Cell·2025
New models for MPNST: establishment and comprehensive characterization of two tumor cell lines
Cancer Cell International·2025· 1 citedOpen Access
Loss of UFMylation supports prostate cancer metastasis and rewires cell metabolism towards hexosamine biosynthesis
2025Open Access
New Models for MPNST: Establishment and Comprehensive Characterization of Two Tumor Cell Lines
2025Open Access
Impact of Low-Dose Computed Tomography Findings on Cigarette Smoking Cessation Among High-Risk Adults Participating in Lung Cancer Screening
Nicotine & Tobacco Research·2025· 1 citedOpen Access
Supplementary figures from Genomic Profiling of Patient-Derived Xenografts for Lung Cancer Identifies <i>B2M</i> Inactivation Impairing Immunorecognition
2025Open Access
Supplementary Table S3 from Genomic Profiling of Patient-Derived Xenografts for Lung Cancer Identifies <i>B2M</i> Inactivation Impairing Immunorecognition
2025Open Access
Supplementary Table 1 from Genomic Profiling of Patient-Derived Xenografts for Lung Cancer Identifies <i>B2M</i> Inactivation Impairing Immunorecognition
2025Open Access
Supplementary Table S2 from Genomic Profiling of Patient-Derived Xenografts for Lung Cancer Identifies <i>B2M</i> Inactivation Impairing Immunorecognition
2025Open Access
Data from Genomic Profiling of Patient-Derived Xenografts for Lung Cancer Identifies <i>B2M</i> Inactivation Impairing Immunorecognition
2025Open Access
Supplementary methods and materials from Genomic Profiling of Patient-Derived Xenografts for Lung Cancer Identifies <i>B2M</i> Inactivation Impairing Immunorecognition
2025Open Access
Legends to supplementary figures and tables from Genomic Profiling of Patient-Derived Xenografts for Lung Cancer Identifies <i>B2M</i> Inactivation Impairing Immunorecognition
2025Open Access
Novel selective strategies targeting the BCL-2 family to enhance clinical efficacy in ALK-rearranged non-small cell lung cancer
Cell Death and Disease·2025· 5 citedOpen Access
Supplementary Figure S6 from Triple Combination of MEK, BET, and CDK Inhibitors Significantly Reduces Human Malignant Peripheral Nerve Sheath Tumors in Mouse Models
2025Open Access
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Frequent Co-authors

Manel Esteller(23), August Vidal(18), Cátia Moutinho(12), Holger Heyn(9), Gabriel Capellá(8), Agustín F. Fernández(7), Sebastián Morán(7), Santiago Ropero(6), Montse Sánchez‐Céspedes(6), F. Javier Carmona(6), Fernando Setién(6), Marta Soler(6), Sònia Guil(6), Anna Martínez‐Cardús(6), Vı́ctor Moreno(5), Ernest Nadal(5), María Martínez‐Iniesta(5), Antonio Gómez(5), Ramón Salazar(5), Manuel Hidalgo(4)