Roger K. Sunahara

University of California, San Diego — United States

Biochemistry, Genetics and Molecular Biology · Life Sciences

63h-index25.7kcitations196works14.52yr avg

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Research Topics

Receptor Mechanisms and Signaling(96), Neurotransmitter Receptor Influence on Behavior(38), Neuropeptides and Animal Physiology(33), Protein Kinase Regulation and GTPase Signaling(27), Forensic Toxicology and Drug Analysis(19)

Publications194 total

Structure and dynamics determine G protein coupling specificity at a class A GPCR

23 cited

Science Advances·2025· 23 citedOpen Access

Structure and dynamics determine G protein coupling specificity at a class A GPCR

6 cited

2024· 6 citedOpen Access

Author Correction: Constrained catecholamines gain β2AR selectivity through allosteric effects on pocket dynamics

Nature Communications·2023Open Access

Constrained catecholamines gain β2AR selectivity through allosteric effects on pocket dynamics

24 cited

Nature Communications·2023· 24 citedOpen Access

Supplementary table from Integrative RNA-omics Discovers GNAS Alternative Splicing as a Phenotypic Driver of Splicing Factor–Mutant Neoplasms

2023Open Access

Supplementary table from Integrative RNA-omics Discovers GNAS Alternative Splicing as a Phenotypic Driver of Splicing Factor–Mutant Neoplasms

2023Open Access

Supplementary table from Integrative RNA-omics Discovers GNAS Alternative Splicing as a Phenotypic Driver of Splicing Factor–Mutant Neoplasms

2023Open Access

Supplementary table from Integrative RNA-omics Discovers GNAS Alternative Splicing as a Phenotypic Driver of Splicing Factor–Mutant Neoplasms

2023Open Access

Supplementary table from Integrative RNA-omics Discovers GNAS Alternative Splicing as a Phenotypic Driver of Splicing Factor–Mutant Neoplasms

2023Open Access

Supplementary table from Integrative RNA-omics Discovers GNAS Alternative Splicing as a Phenotypic Driver of Splicing Factor–Mutant Neoplasms

2023Open Access

Supplementary table from Integrative RNA-omics Discovers GNAS Alternative Splicing as a Phenotypic Driver of Splicing Factor–Mutant Neoplasms

2023Open Access

Supplementary table from Integrative RNA-omics Discovers GNAS Alternative Splicing as a Phenotypic Driver of Splicing Factor–Mutant Neoplasms

2023Open Access

Supplementary table from Integrative RNA-omics Discovers GNAS Alternative Splicing as a Phenotypic Driver of Splicing Factor–Mutant Neoplasms

2023Open Access

Supplementary table from Integrative RNA-omics Discovers GNAS Alternative Splicing as a Phenotypic Driver of Splicing Factor–Mutant Neoplasms

2023Open Access

Supplementary Data from Integrative RNA-omics Discovers GNAS Alternative Splicing as a Phenotypic Driver of Splicing Factor–Mutant Neoplasms

2023Open Access

Supplementary Data from Integrative RNA-omics Discovers GNAS Alternative Splicing as a Phenotypic Driver of Splicing Factor–Mutant Neoplasms

2023Open Access

Data from Integrative RNA-omics Discovers GNAS Alternative Splicing as a Phenotypic Driver of Splicing Factor–Mutant Neoplasms

2023Open Access

Supplementary table from Integrative RNA-omics Discovers GNAS Alternative Splicing as a Phenotypic Driver of Splicing Factor–Mutant Neoplasms

2023Open Access

Supplementary table from Integrative RNA-omics Discovers GNAS Alternative Splicing as a Phenotypic Driver of Splicing Factor–Mutant Neoplasms

2023Open Access

Data from Integrative RNA-omics Discovers GNAS Alternative Splicing as a Phenotypic Driver of Splicing Factor–Mutant Neoplasms

2023Open Access

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Frequent Co-authors

Brian K. Kobilka(13), Hyman B. Niznik(10), Søren G. F. Rasmussen(9), Brian T. DeVree(9), Jan Steyaert(7), Philip Seeman(7), Alfred G. Gilman(7), Jacob P. Mahoney(6), Pil Seok Chae(5), Els Pardon(5), William I. Weis(5), Brian F. O’Dowd(5), J.J.G. Tesmer(5), Stephen R. Sprang(5), Matthew R. Whorton(4), Gisselle A. Vélez-Ruiz(4), Ka Young Chung(3), Tong Sun Kobilka(3), Foon Sun Thian(3), Diane Calinski(3)