Julia Stindl

Novocure (Jersey) — JE

Medicine · Health Sciences

11h-index697citations21works0.02yr avg
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Research Topics

Hormonal Regulation and Hypertension(10), Ion Transport and Channel Regulation(8), Retinal Development and Disorders(4), Glioma Diagnosis and Treatment(3), Renin-Angiotensin System Studies(3)

Publications21 total

The safety profile of Tumor Treating Fields (TTFields) therapy in glioblastoma patients with ventriculoperitoneal shunts
Journal of Neuro-Oncology·2022· 10 citedOpen Access
Feasibility and safety of TTFields therapy for glioblastoma patients with ventriculoperitoneal shunt
WFNOS 2022 Abstract Book·2022Open Access
SURG-06. THE SAFETY PROFILE OF TUMOR TREATING FIELDS (TTFIELDS; 200 KHZ) CONCOMITANT WITH VENTRICULO-PERITONEAL SHUNTS IN PATIENTS WITH GLIOBLASTOMA AND HYDROCEPHALUS
Neuro-Oncology·2021Open Access
Cellular Pathophysiology of Mutant Voltage-Dependent Ca2+ Channel CACNA1H in Primary Aldosteronism
Endocrinology·2020· 13 citedOpen Access
Systemic ß adrenergic stimulation/ sympathetic nerve system stimulation influences intraocular RAS through cAMP in the RPE
Experimental Eye Research·2019· 7 cited
OS5.5 Surveillance data demonstrates the tolerability of tumor treating fields in pediatric glioma patients
Neuro-Oncology·2018· 1 citedOpen Access
Cellular Pathophysiology of an Adrenal Adenoma-Associated Mutant of the Plasma Membrane Ca2+-ATPase ATP2B3
Endocrinology·2016· 66 citedOpen Access
Untersuchungen zur Entstehung des primären Hyperaldosteronismus: Einfluss von Mutationen der Na+/K+-ATPase auf adrenokortikale Zellen
University of Regensburg Publication Server (University of Regensburg)·2016Open Access
Pathogenesis of Adrenal Aldosterone-Producing Adenomas Carrying Mutations of the Na+/K+-ATPase
Endocrinology·2015· 62 citedOpen Access
6C.03
Journal of Hypertension·2015
Oxytocin Stimulates Extracellular Ca2+ Influx Through TRPV2 Channels in Hypothalamic Neurons to Exert Its Anxiolytic Effects
Neuropsychopharmacology·2015· 63 citedOpen Access
Pathophysiology of Na+/K+-atpases in aldosterone secretion
Experimental and Clinical Endocrinology & Diabetes·2015
Activation of endogenously expressed ion channels by active complement in the retinal pigment epithelium
Pflügers Archiv - European Journal of Physiology·2014· 17 cited
A Case of Severe Hyperaldosteronism Caused by a De Novo Mutation Affecting a Critical Salt Bridge Kir3.4 Residue
The Journal of Clinical Endocrinology & Metabolism·2014· 57 citedOpen Access
Abstract 011: Identification and Electrophysiological Characterization of a Novel Somatic Mutation (insT149KCNJ5) of the Potassium Channel Kir3.4 (KCNJ5)
Hypertension·2014
A Novel KCNJ5-insT149 Somatic Mutation Close to, but Outside, the Selectivity Filter Causes Resistant Hypertension by Loss of Selectivity for Potassium
The Journal of Clinical Endocrinology & Metabolism·2014· 57 citedOpen Access
Pharmacology and Pathophysiology of Mutated KCNJ5 Found in Adrenal Aldosterone-Producing Adenomas
Endocrinology·2014· 65 citedOpen Access
Somatic <i>ATP1A1</i> , <i>ATP2B3</i> , and <i>KCNJ5</i> Mutations in Aldosterone-Producing Adenomas
Hypertension·2013· 164 citedOpen Access
Activation of the retinal renin angiotensin system via beta-adrenergic stimulation of renin expression in the RPE
Investigative Ophthalmology & Visual Science·2013· 1 cited
Angiotensin-2-Mediated Ca2+ Signaling in the Retinal Pigment Epithelium: Role of Angiotensin-Receptor- Associated-Protein and TRPV2 Channel
PLoS ONE·2012· 48 citedOpen Access
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Frequent Co-authors

Sascha Bandulik(9), Richard Warth(8), Olaf Strauß(6), Philipp Tauber(4), Ines Tegtmeier(4), Tracy Ann Williams(3), Silvia Monticone(3), Felix Beuschlein(3), Paolo Mulatero(3), Christina Sterner(3), Vladimir Todorov(3), Nancy Ann Oberheim-Bush(3), Wenyin Shi(3), Alexander Grote(3), Leonardo Lustgarten(3), Martín Reincke(2), Mihail Zilbermint(2), И. И. Дедов(2), Michael Allgäeuer(2), Chyi‐Chia Richard Lee(2)