Meike Hohwieler

Universität Ulm — Germany

Medicine · Health Sciences

16h-index993citations72works0.62yr avg
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Research Topics

Pancreatic and Hepatic Oncology Research(29), Pancreatic function and diabetes(25), Cancer Genomics and Diagnostics(14), Renal and related cancers(13), Pluripotent Stem Cells Research(11)

Publications72 total

An oncogenic KRAS-driven secretome involving TNFα promotes niche preparation prior to pancreatic cancer onset
Molecular Cancer·2026Open Access
An oncogenic KRAS-driven secretome involving TNFα promotes niche preparation prior to pancreatic cancer onset
Figshare·2026Open Access
Additional file 1 of An oncogenic KRAS-driven secretome involving TNFα promotes niche preparation prior to pancreatic cancer onset
Figshare·2026Open Access
An oncogenic KRAS-driven secretome involving TNFα promotes niche preparation prior to pancreatic cancer onset
Figshare·2026Open Access
Additional file 1 of An oncogenic KRAS-driven secretome involving TNFα promotes niche preparation prior to pancreatic cancer onset
Figshare·2026Open Access
Oncogenic GNAS and KRAS signaling collectively drive dysplasia in an iPSC-derived organoid model for cystic pancreatic neoplasia
Pancreatology·2025
Novel ploidy-control genes ORP3 and GJB3 link aneuploidy to tumor cell invasion through microtubule and actin dynamics
2025Open Access
PPDPF is not a key regulator of human pancreas development
PLoS Genetics·2025Open Access
A ONECUT1 regulatory, non-coding region in pancreatic development and diabetes
Cell Reports·2024· 2 citedOpen Access
Abstract C010: Single-cell profiling unleashes KRAS-driven redrafting of the tumor microenvironment before cancer onset
Cancer Research·2024
Single-cell profiling unleashes KRAS-driven redrafting of the tumor microenvironment before cancer onset
Zeitschrift für Gastroenterologie·2024
A ONECUT1 regulatory, non-coding region in pancreatic development and diabetes
2024· 1 citedOpen Access
Impairment of α-tubulin and F-actin interactions of GJB3 induces aneuploidy in urothelial cells and promotes bladder cancer cell invasion
Cellular & Molecular Biology Letters·2024· 5 citedOpen Access
Allelic regulation of Keratin 19 gene expression during pancreatic development and carcinogenesis
Pancreatology·2023
Single-cell profiling of GP2-enriched pancreatic progenitors to simultaneously create acinar, ductal and endocrine organoids
Pancreatology·2023
Loss of ORP3 induces aneuploidy and promotes bladder cancer cell invasion through deregulated microtubule and actin dynamics
Cellular and Molecular Life Sciences·2023· 9 citedOpen Access
<scp>DNA</scp> methylation‐associated allelic inactivation regulates Keratin 19 gene expression during pancreatic development and carcinogenesis
The Journal of Pathology·2023· 6 citedOpen Access
Figure S1 GSEA Analysis from ATM Deficiency Generating Genomic Instability Sensitizes Pancreatic Ductal Adenocarcinoma Cells to Therapy-Induced DNA Damage
2023Open Access
Figure S2 from ATM Deficiency Generating Genomic Instability Sensitizes Pancreatic Ductal Adenocarcinoma Cells to Therapy-Induced DNA Damage
2023Open Access
Figure S4 Tumour growth upon ATRi and Gemcitabine Treatment in Allografts model from ATM Deficiency Generating Genomic Instability Sensitizes Pancreatic Ductal Adenocarcinoma Cells to Therapy-Induced DNA Damage
2023Open Access
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Frequent Co-authors

Alexander Kleger(46), Thomas Seufferlein(27), Markus Breunig(26), Stefan Liebau(16), Sandra Heller(16), Lukas Perkhofer(15), Ninel Azoitei(13), André Lechel(11), Jessica Merkle(11), Ronan Russell(10), Anett Illing(9), Martin C. Müller(9), Jana Krüger(9), Ivan G. Costa(8), Maria Carolina Romero Carrasco(8), Bence Sipos(8), Pierre‐Olivier Frappart(8), Michael Karl Melzer(8), Christian Bolenz(8), Chantal Allgöwer(8)