Vito Guagnano

Biochemistry, Genetics and Molecular Biology · Life Sciences

19h-index2.3kcitations74works0.02yr avg
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Research Topics

Fibroblast Growth Factor Research(20), Synthetic Organic Chemistry Methods(10), Cancer-related Molecular Pathways(6), Epigenetics and DNA Methylation(5), Cancer, Hypoxia, and Metabolism(5)

Publications74 total

Data from FGFR Genetic Alterations Predict for Sensitivity to NVP-BGJ398, a Selective Pan-FGFR Inhibitor
2023Open Access
Supplementary Table 5 from FGFR Genetic Alterations Predict for Sensitivity to NVP-BGJ398, a Selective Pan-FGFR Inhibitor
2023Open Access
Data from FGFR Genetic Alterations Predict for Sensitivity to NVP-BGJ398, a Selective Pan-FGFR Inhibitor
2023Open Access
Supplementary Figures 1-5 from Rescue Screens with Secreted Proteins Reveal Compensatory Potential of Receptor Tyrosine Kinases in Driving Cancer Growth
2023Open Access
Supplementary Tables 1-3 and Table 5 from Rescue Screens with Secreted Proteins Reveal Compensatory Potential of Receptor Tyrosine Kinases in Driving Cancer Growth
2023Open Access
Data from Rescue Screens with Secreted Proteins Reveal Compensatory Potential of Receptor Tyrosine Kinases in Driving Cancer Growth
2023Open Access
Supplementary Figure Legends 1-5 from FGFR Genetic Alterations Predict for Sensitivity to NVP-BGJ398, a Selective Pan-FGFR Inhibitor
2023Open Access
Supplementary Table 5 from FGFR Genetic Alterations Predict for Sensitivity to NVP-BGJ398, a Selective Pan-FGFR Inhibitor
2023Open Access
Supplementary Tables 1-3 and Table 5 from Rescue Screens with Secreted Proteins Reveal Compensatory Potential of Receptor Tyrosine Kinases in Driving Cancer Growth
2023Open Access
Supplementary Tables 1-4 from FGFR Genetic Alterations Predict for Sensitivity to NVP-BGJ398, a Selective Pan-FGFR Inhibitor
2023Open Access
Supplementary Figures 1-5 from Rescue Screens with Secreted Proteins Reveal Compensatory Potential of Receptor Tyrosine Kinases in Driving Cancer Growth
2023Open Access
Supplementary Table 4 from Rescue Screens with Secreted Proteins Reveal Compensatory Potential of Receptor Tyrosine Kinases in Driving Cancer Growth
2023Open Access
Supplementary Tables 1-4 from FGFR Genetic Alterations Predict for Sensitivity to NVP-BGJ398, a Selective Pan-FGFR Inhibitor
2023Open Access
Supplementary Methods from Rescue Screens with Secreted Proteins Reveal Compensatory Potential of Receptor Tyrosine Kinases in Driving Cancer Growth
2023Open Access
Supplementary Methods from Rescue Screens with Secreted Proteins Reveal Compensatory Potential of Receptor Tyrosine Kinases in Driving Cancer Growth
2023Open Access
Data from Rescue Screens with Secreted Proteins Reveal Compensatory Potential of Receptor Tyrosine Kinases in Driving Cancer Growth
2023Open Access
Supplementary Figures 1-5 from FGFR Genetic Alterations Predict for Sensitivity to NVP-BGJ398, a Selective Pan-FGFR Inhibitor
2023Open Access
Supplementary Figures 1-5 from FGFR Genetic Alterations Predict for Sensitivity to NVP-BGJ398, a Selective Pan-FGFR Inhibitor
2023Open Access
Supplementary Table 4 from Rescue Screens with Secreted Proteins Reveal Compensatory Potential of Receptor Tyrosine Kinases in Driving Cancer Growth
2023Open Access
Supplementary Figure Legends 1-5 from FGFR Genetic Alterations Predict for Sensitivity to NVP-BGJ398, a Selective Pan-FGFR Inhibitor
2023Open Access
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Frequent Co-authors

Pascal Furet(16), Francesco Hofmann(15), Christelle Stamm(13), William R. Sellers(12), Vincent Bordas(10), Mickaël Le Douget(10), Diana Graus-Porta(9), Joerg Kallen(8), Andrea Vaupel(8), Rita Menicagli(8), Giorgio Caravatti(7), Audrey Kauffmann(7), Simon Wöhrle(7), Moriko Ito(7), Thomas Brümmendorf(7), Stefan Stutz(7), Patrick Chêne(7), Scott E. Denmark(7), Markus Wartmann(6), Astrid Pornon(6)