Masaharu Takigawa

Okayama University — Japan

Biochemistry, Genetics and Molecular Biology · Life Sciences

77h-index19.9kcitations622works1.02yr avg
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Research Topics

Connective Tissue Growth Factor Research(240), Osteoarthritis Treatment and Mechanisms(89), Connective tissue disorders research(42), Fibroblast Growth Factor Research(38), Cell Adhesion Molecules Research(32)

Publications618 total

Interaction between nuclear‐translocated cellular communication network factor 2 and purine‐rich box 1 regulates the expression of fibrosis‐related genes
Journal of Cell Communication and Signaling·2025· 1 citedOpen Access
Estimating influenza vaccine effectiveness among older adults using an integrated administrative database and the implications of potential bias: A population-based cohort study in Japan
Vaccine·2024· 1 cited
Positive Regulation of S-Adenosylmethionine on Chondrocytic Differentiation via Stimulation of Polyamine Production and the Gene Expression of Chondrogenic Differentiation Factors
International Journal of Molecular Sciences·2023· 2 citedOpen Access
Positive Regulation of S-adenosylmethionine on Chondrocytic Differentiation via Stimulation of Polyamine Production and the Gene Expression of Chondrogenic Differentiation Factors
Preprints.org·2023· 1 citedOpen Access
Expression and function of CCN2-derived circRNAs in chondrocytes
Journal of Cell Communication and Signaling·2023Open Access
Dual roles of cellular communication network factor 6 (CCN6) in the invasion and metastasis of oral cancer cells to bone via binding to BMP2 and RANKL
Carcinogenesis·2023· 1 citedOpen Access
Supplementary Figure Legends 1-7 from Proteasome Inhibition Up-regulates p53 and Apoptosis-Inducing Factor in Chondrocytes Causing Severe Growth Retardation in Mice
2023Open Access
Data from Proteasome Inhibition Up-regulates p53 and Apoptosis-Inducing Factor in Chondrocytes Causing Severe Growth Retardation in Mice
2023Open Access
Supplementary Figure Legends 1-7 from Proteasome Inhibition Up-regulates p53 and Apoptosis-Inducing Factor in Chondrocytes Causing Severe Growth Retardation in Mice
2023Open Access
Supplementary Figures 1-7 from Proteasome Inhibition Up-regulates p53 and Apoptosis-Inducing Factor in Chondrocytes Causing Severe Growth Retardation in Mice
2023Open Access
Data from Proteasome Inhibition Up-regulates p53 and Apoptosis-Inducing Factor in Chondrocytes Causing Severe Growth Retardation in Mice
2023Open Access
Supplementary Figures 1-7 from Proteasome Inhibition Up-regulates p53 and Apoptosis-Inducing Factor in Chondrocytes Causing Severe Growth Retardation in Mice
2023Open Access
Do not overwork: cellular communication network factor 3 for life in cartilage
Journal of Cell Communication and Signaling·2023· 6 citedOpen Access
Elevated Expression of CCN3 in Articular Cartilage Induces Osteoarthritis in Hip Joints Irrespective of Age and Weight Bearing
International Journal of Molecular Sciences·2022· 8 citedOpen Access
The Evaluation of Meniscus Regenerative Effects of LIPUS-Induced CCN Proteins: Induction by LIPUS of CCN2 and Meniscus-Related Genes in Cultured Meniscus Cells and Meniscus Tissues
Methods in molecular biology·2022· 3 cited
Evaluation of the Molecular Interaction Between CCN Protein and Its Binding Partners: A Solid-Phase Binding Assay and Surface Plasmon Resonance
Methods in molecular biology·2022· 2 cited
Imaging of Molecular Interaction Between CCN Protein and Its Binding Partners: An In Situ Proximity Ligation Assay of Interaction Between CCN2 and Rab14 in Chondrocytes
Methods in molecular biology·2022
Cellular Fluorescence Imaging for the Evaluation of Bioactivity of CCN Family Proteins
Methods in molecular biology·2022
CCN Proteins (Cellular Communication Network Factors): Expanding Their Repertoire Toward a New Concept
Methods in molecular biology·2022· 7 cited
Protocols for Screening Peptides Binding to CCN Family Proteins and Their Extended Utility
Methods in molecular biology·2022
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Frequent Co-authors

Tohru Nakanishi(13), Takashi Nishida(13), Satoshi Kubota(12), Tsuyoshi Shimo(6), Takuo Kuboki(5), Takuya Tamatani(5), Toru Nakanishi(4), Katsunari Tezuka(4), Takanori Eguchi(4), Takako Hattori(4), Kazuhiko Takehara(3), Masahiro Asano(3), Tetsuo Hattori(3), Soichiro Ibaragi(3), Akira Sasaki(3), Gen Yosimichi(3), Toshikazu Kubo(2), Shigeru Kawara(2), Takuhiro Matsumura(2), Kazuhiro Sasaki(2)