Eric Seidel

University of Cologne — Germany

Medicine · Health Sciences

11h-index478citations24works6.02yr avg
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Research Topics

Hormonal Regulation and Hypertension(11), Ion Transport and Channel Regulation(5), Renin-Angiotensin System Studies(5), Receptor Mechanisms and Signaling(4), S100 Proteins and Annexins(4)

Publications24 total

An integrated model of population growth saturation and basal ROS levels predicts cellular ferroptosis sensitivity
2025Open Access
An atlas of ferroptosis-induced secretomes
Cell Death and Differentiation·2025· 12 citedOpen Access
Isradipine therapy in Cacna1dIle772Met/+ mice ameliorates primary aldosteronism and neurologic abnormalities
JCI Insight·2023· 13 citedOpen Access
Dataset to: Isradipine therapy in Cacna1dIle772Met/+ mice with PASNA syndrome (primary aldosteronism, seizures and neurologic abnormalities)
2023Open Access
Dataset to: Isradipine therapy in Cacna1dIle772Met/+ mice with PASNA syndrome (primary aldosteronism, seizures and neurologic abnormalities)
Zenodo (CERN European Organization for Nuclear Research)·2023Open Access
Elevated FSP1 protects KRAS-mutated cells from ferroptosis during tumor initiation
Cell Death and Differentiation·2022· 117 citedOpen Access
Extrinsic cell death pathway plasticity: a driver of clonal evolution in cancer?
Cell Death Discovery·2022· 6 citedOpen Access
Author Correction: Elevated aldosterone and blood pressure in a mouse model of familial hyperaldosteronism with ClC-2 mutation
Nature Communications·2022· 1 citedOpen Access
Dual roles of B lymphocytes in mouse models of diet‐induced nonalcoholic fatty liver disease
Hepatology·2022· 37 citedOpen Access
BP telemetry data for Seidel et al., PNAS, 2021
2021Open Access
BP telemetry data for Seidel et al., PNAS, 2021
Zenodo (CERN European Organization for Nuclear Research)·2021Open Access
Enhanced Ca<sup>2+</sup>signaling, mild primary aldosteronism, and hypertension in a familial hyperaldosteronism mouse model (<i>Cacna1h</i><sup><i>M1560V/+</i></sup>)
Proceedings of the National Academy of Sciences·2021· 35 citedOpen Access
Enhanced Ca2+ signaling, mild primary aldosteronism, and hypertension in a familial hyperaldosteronism mouse model (Cacna1h M1560V/+)
2021· 2 citedOpen Access
Enhanced Ca2+ signaling, mild primary aldosteronism, and hypertension in a familial hyperaldosteronism mouse model (Cacna1h M1560V/+)
Zenodo (CERN European Organization for Nuclear Research)·2021Open Access
Retraction notice to “Peptide-induced formation of protein aggregates and amyloid fibrils in human and Guinea pig αA-crystallins under physiological conditions of temperature and pH” [Exp. Eye Res. 179 (2019) 193–205]
Experimental Eye Research·2020· 1 citedOpen Access
Generation and characterization of a mitotane-resistant adrenocortical cell line
Endocrine Connections·2020· 17 citedOpen Access
Elevated aldosterone and blood pressure in a mouse model of familial hyperaldosteronism with ClC-2 mutation
Nature Communications·2019· 49 citedOpen Access
Genetic causes of primary aldosteronism
Experimental & Molecular Medicine·2019· 54 citedOpen Access
RETRACTED: Peptide-induced formation of protein aggregates and amyloid fibrils in human and guinea pig αA-crystallins under physiological conditions of temperature and pH
Experimental Eye Research·2018· 5 citedOpen Access
TEM analysis of αA66-80 peptide-induced protein aggregates and amyloid fibrils in human and guinea pig αA-crystallins
2018· 1 cited
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Frequent Co-authors

Ute I. Scholl(15), Julia Schewe(11), Gabriel Stölting(10), Junhui Zhang(8), Nicole Hellmig(8), Richard P. Lifton(8), Timothy Nottoli(8), Sofia K. Forslund(7), Jörg Peters(7), Dominik N. Müller(7), Hoang An Dinh(6), Silvia von Karstedt(4), Fatma Isil Yapici(3), Lajos Markó(3), Marina Volkert(3), Luise Hennicke(3), Herbert Oberacher(3), Iris Urban(3), Melissa Long(3), Marion Rivalan(3)