Matthew D. Petroski

Pfizer (United States) — United States

Biochemistry, Genetics and Molecular Biology · Life Sciences

21h-index5.0kcitations42works6.12yr avg
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Research Topics

Ubiquitin and proteasome pathways(19), Cancer-related Molecular Pathways(9), Endoplasmic Reticulum Stress and Disease(8), Advanced Breast Cancer Therapies(8), Autophagy in Disease and Therapy(7)

Publications42 total

Supplementary Figure 2 from <sup>18</sup>F-FLT PET, a Noninvasive Pharmacodynamic Biomarker of Tumor Cell Proliferation, Detected Differential Response to Various Cyclin-Dependent Kinase (CDK) Inhibitors
2025Open Access
Supplementary Figure 3 from <sup>18</sup>F-FLT PET, a Noninvasive Pharmacodynamic Biomarker of Tumor Cell Proliferation, Detected Differential Response to Various Cyclin-Dependent Kinase (CDK) Inhibitors
2025Open Access
Supplementary Tables from <sup>18</sup>F-FLT PET, a Noninvasive Pharmacodynamic Biomarker of Tumor Cell Proliferation, Detected Differential Response to Various Cyclin-Dependent Kinase (CDK) Inhibitors
2025Open Access
Data from <sup>18</sup>F-FLT PET, a Noninvasive Pharmacodynamic Biomarker of Tumor Cell Proliferation, Detected Differential Response to Various Cyclin-Dependent Kinase (CDK) Inhibitors
2025Open Access
Supplementary Figure 4 from <sup>18</sup>F-FLT PET, a Noninvasive Pharmacodynamic Biomarker of Tumor Cell Proliferation, Detected Differential Response to Various Cyclin-Dependent Kinase (CDK) Inhibitors
2025Open Access
Supplementary Figure 1 from <sup>18</sup>F-FLT PET, a Noninvasive Pharmacodynamic Biomarker of Tumor Cell Proliferation, Detected Differential Response to Various Cyclin-Dependent Kinase (CDK) Inhibitors
2025Open Access
18F-FLT PET, a Noninvasive Pharmacodynamic Biomarker of Tumor Cell Proliferation, Detected Differential Response to Various Cyclin-Dependent Kinase (CDK) Inhibitors
Molecular Cancer Therapeutics·2025· 3 citedOpen Access
CDK4 selective inhibition improves preclinical anti-tumor efficacy and safety
Cancer Cell·2025· 36 citedOpen Access
An allosteric cyclin E-CDK2 site mapped by paralog hopping with covalent probes
Nature Chemical Biology·2024· 15 citedOpen Access
Expanding the ligandable proteome by paralog hopping with covalent probes
2024· 7 citedOpen Access
Phosphorylation of SAMHD1 Thr592 increases C-terminal domain dynamics, tetramer dissociation and ssDNA binding kinetics
Nucleic Acids Research·2022· 15 citedOpen Access
Phosphorylation of SAMHD1 Thr592 increases C-terminal domain dynamics, tetramer dissociation, and ssDNA binding kinetics
2022Open Access
Adapted ATPase domain communication overcomes the cytotoxicity of p97 inhibitors
Journal of Biological Chemistry·2018· 13 citedOpen Access
p97 Composition Changes Caused by Allosteric Inhibition Are Suppressed by an On-Target Mechanism that Increases the Enzyme's ATPase Activity
Cell chemical biology·2016· 42 citedOpen Access
Mutations in UBA3 Confer Resistance to the NEDD8-Activating Enzyme Inhibitor MLN4924 in Human Leukemic Cells
PLoS ONE·2014· 42 citedOpen Access
Lys11- and Lys48-linked ubiquitin chains interact with p97 during endoplasmic-reticulum-associated degradation
Biochemical Journal·2014· 60 citedOpen Access
N6-methyladenosine modification destabilizes developmental regulators in embryonic stem cells
Nature Cell Biology·2014· 1.3k cited
developmental regulators in embryonic stem cells
2014
Mutations In UBA3 Confer Resistance To The NEDD8-Activating Enzyme Inhibitor MLN4924 In Human Leukemic Cells
Blood·2013· 9 cited
The Cyclomodulin Cycle Inhibiting Factor (CIF) Alters Cullin Neddylation Dynamics
Journal of Biological Chemistry·2013· 21 citedOpen Access
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Frequent Co-authors

Julia I. Toth(13), Jason Carmody(10), Todd VanArsdale(10), Britton Boras(8), Nanni Huser(8), Stephen G. Dann(8), Anand Giddabasappa(7), Ziyue Karen Jiang(7), Bing Yang(7), Laigao Chen(7), Feng Liu(7), Edward Cabral(7), Sripad Ram(7), Cathy C. Zhang(7), Kavon Noorbehesht(7), Lisa K. Manzuk(7), Ravi Visswanathan(7), Sepideh Mojtahedzadeh(7), Timothy Affolter(7), Quang‐Dé Nguyen(7)