Jonathan G. Seidman

Harvard University — United States

Medicine · Health Sciences

132h-index92.1kcitations532works7.52yr avg
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Research Topics

Cardiomyopathy and Myosin Studies(190), Congenital heart defects research(103), Cardiovascular Effects of Exercise(56), Viral Infections and Immunology Research(54), Muscle Physiology and Disorders(40)

Publications493 total

Abstract 4363833: Impact of Damaging Genetic Variants on Right Ventricular Function in Tetralogy of Fallot
Circulation·2025
Abstract 4371113: Differential Gene Expression Signatures Underlying Postoperative Atrial Fibrillation
Circulation·2025
Abstract 4344599: Converging Mechanisms in Divergent Cardiomyopathies: FSCN1 as a Druggable Epigenetic-Mitochondrial Target in Heart Failure
Circulation·2025
Abstract 4339547: Nicotinamide Nucleoside Transhydrogenase (NNT) loss-of-function attenuates oxidative stress-associated myocardial fibrosis and diastolic dysfunction in cardiometabolic HFpEF
Circulation·2025
Thick filament molecular interfaces play a critical role in pathogenesis of hypertrophic and dilated cardiomyopathy
2025· 2 citedOpen Access
Reversible compromise of physiological resilience by accumulation of heteroplasmic mtDNA mutations
Science·2025· 3 citedOpen Access
Endocardial primary cilia and blood flow regulate EndoMT during endocardial cushion development
Nature Cardiovascular Research·2025Open Access
Abstract NBT201: Genetically Barcoded Multiplex Single Cell Analysis of Variants in Congenital Heart Disease
Circulation Research·2025
Enrichment of tandem repeat element variants near CHD genes identified by short- and long-read genome sequencing
BMC Medical Genomics·2025Open Access
Genome sequencing is critical for forecasting outcomes following congenital cardiac surgery
Nature Communications·2025· 1 citedOpen Access
Regulatory T cells attenuate chronic inflammation and cardiac fibrosis in hypertrophic cardiomyopathy
Science Translational Medicine·2025· 6 cited
Mitochondrial NNT Promotes Diastolic Dysfunction in Cardiometabolic HFpEF
Circulation Research·2025· 13 cited
Empagliflozin enhances metabolic efficiency and improves left ventricular hypertrophy in a hypertrophic cardiomyopathy mouse model
European Heart Journal·2025· 7 citedOpen Access
BMAL1–HIF2A heterodimer modulates circadian variations of myocardial injury
Nature·2025· 32 citedOpen Access
Genomic analysis of 11,555 probands identifies 60 dominant congenital heart disease genes
Proceedings of the National Academy of Sciences·2025· 15 citedOpen Access
Recessive genetic contribution to congenital heart disease in 5,424 probands
Proceedings of the National Academy of Sciences·2025· 9 citedOpen Access
Modeling <i>SMAD2</i> Mutations in Induced Pluripotent Stem Cells Provides Insights Into Cardiovascular Disease Pathogenesis
Journal of the American Heart Association·2025· 3 citedOpen Access
The cellular and molecular cardiac tissue responses in human inflammatory cardiomyopathies after SARS-CoV-2 infection and COVID-19 vaccination
Nature Cardiovascular Research·2025· 11 citedOpen Access
Genes of the Fatty Acid Oxidation Pathway are Upregulated in Female as Compared to Male Cardiomyocytes
Circulation·2025· 2 cited
Contribution of hypoxia-inducible factor 1alpha to pathogenesis of sarcomeric hypertrophic cardiomyopathy
Scientific Reports·2025· 2 citedOpen Access
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Frequent Co-authors

Christine E. Seidman(38), Hiroko Wakimoto(11), Joshua Gorham(9), Stuart A. Cook(8), Steven R. DePalma(7), William J. McKenna(6), Raju Kucherlapati(5), Norbert Hübner(5), Paul J.R. Barton(5), James S. Ware(5), Lynda Chin(4), Angela Hadjipanayis(4), Semin Lee(4), Peter J. Park(4), Lixing Yang(4), Barbara McDonough(4), Carolyn Y. Ho(4), Benoit G. Bruneau(4), David A. Conner(4), Amy E. Roberts(4)