William Clark

Medicine · Health Sciences

49h-index8.2kcitations264works4.12yr avg
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Research Topics

Pancreatic and Hepatic Oncology Research(16), Epigenetics and DNA Methylation(15), Cardiomyopathy and Myosin Studies(13), Diet and metabolism studies(12), Phagocytosis and Immune Regulation(12)

Publications264 total

Onset of millennial climate variability with the intensification of Northern Hemisphere glaciation.
Apollo (University of Cambridge)·2026Open Access
Uridine phosphorylase-1 supports metastasis by altering immune and extracellular matrix landscapes
EMBO Reports·2025· 3 citedOpen Access
Effects of Maternal Pre-Pregnancy BMI on Preterm Infant Microbiome and Fecal Fermentation Profile—A Preliminary Cohort Study
Nutrients·2025· 1 citedOpen Access
Supplementary Figure 2 from <i>KRAS</i> Loss of Heterozygosity Promotes MAPK-Dependent Pancreatic Ductal Adenocarcinoma Initiation and Induces Therapeutic Sensitivity to MEK Inhibition
2025Open Access
Supplementary Figure 4 from <i>KRAS</i> Loss of Heterozygosity Promotes MAPK-Dependent Pancreatic Ductal Adenocarcinoma Initiation and Induces Therapeutic Sensitivity to MEK Inhibition
2025Open Access
Supplementary Figure 3 from <i>KRAS</i> Loss of Heterozygosity Promotes MAPK-Dependent Pancreatic Ductal Adenocarcinoma Initiation and Induces Therapeutic Sensitivity to MEK Inhibition
2025Open Access
Figure 1 from <i>KRAS</i> Loss of Heterozygosity Promotes MAPK-Dependent Pancreatic Ductal Adenocarcinoma Initiation and Induces Therapeutic Sensitivity to MEK Inhibition
2025Open Access
Data from <i>KRAS</i> Loss of Heterozygosity Promotes MAPK-Dependent Pancreatic Ductal Adenocarcinoma Initiation and Induces Therapeutic Sensitivity to MEK Inhibition
2025Open Access
Supplementary Figure 1 from <i>KRAS</i> Loss of Heterozygosity Promotes MAPK-Dependent Pancreatic Ductal Adenocarcinoma Initiation and Induces Therapeutic Sensitivity to MEK Inhibition
2025Open Access
Supplementary Figure 5 from <i>KRAS</i> Loss of Heterozygosity Promotes MAPK-Dependent Pancreatic Ductal Adenocarcinoma Initiation and Induces Therapeutic Sensitivity to MEK Inhibition
2025Open Access
Figure 4 from <i>KRAS</i> Loss of Heterozygosity Promotes MAPK-Dependent Pancreatic Ductal Adenocarcinoma Initiation and Induces Therapeutic Sensitivity to MEK Inhibition
2025Open Access
Supplementary Figure 6 from <i>KRAS</i> Loss of Heterozygosity Promotes MAPK-Dependent Pancreatic Ductal Adenocarcinoma Initiation and Induces Therapeutic Sensitivity to MEK Inhibition
2025Open Access
Figure 2 from <i>KRAS</i> Loss of Heterozygosity Promotes MAPK-Dependent Pancreatic Ductal Adenocarcinoma Initiation and Induces Therapeutic Sensitivity to MEK Inhibition
2025Open Access
Figure 3 from <i>KRAS</i> Loss of Heterozygosity Promotes MAPK-Dependent Pancreatic Ductal Adenocarcinoma Initiation and Induces Therapeutic Sensitivity to MEK Inhibition
2025Open Access
Hepatocellular senescence induces multi-organ senescence and dysfunction via TGFβ
Nature Cell Biology·2024· 44 citedOpen Access
<i>KRAS</i> Loss of Heterozygosity Promotes MAPK-Dependent Pancreatic Ductal Adenocarcinoma Initiation and Induces Therapeutic Sensitivity to MEK Inhibition
Cancer Research·2024· 6 citedOpen Access
Inflammatory markers and body mass index amoung hispanic children
PLoS ONE·2024· 5 citedOpen Access
KRAS allelic imbalance drives tumour initiation yet suppresses metastasis in colorectal cancer in vivo
Nature Communications·2024· 15 citedOpen Access
Stay or go? Most older Australians want to retire where they are, but renters don’t always get a choice
2023
<i>KRAS</i> allelic imbalance drives tumour initiation yet suppresses metastasis in colorectal cancer <i>in vivo</i>
2023Open Access
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Frequent Co-authors

Colin Nixon(18), Owen J. Sansom(12), Ann Hedley(9), Rachel A. Ridgway(8), Andrew D. Campbell(7), Simon T. Barry(7), Kathryn Gilroy(7), Peter D. Adams(7), Joshua D.G. Leach(6), R Zak(6), John Cole(6), Jennifer P. Morton(5), Thomas G. Bird(5), René Jackstadt(4), Arafath K. Najumudeen(4), Neil Robertson(4), Tony McBryan(4), Karen Blyth(4), Andrew V. Biankin(3), Nikola Vlahov(3)