Stephen J. Kron

University of Chicago — United States

Biochemistry, Genetics and Molecular Biology · Life Sciences

53h-index11.1kcitations322works2.02yr avg
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Research Topics

DNA Repair Mechanisms(43), Cancer Research and Treatments(34), Cancer, Hypoxia, and Metabolism(28), Fungal and yeast genetics research(27), Telomeres, Telomerase, and Senescence(27)

Publications322 total

A Non-Invasive Path to Animal Welfare: Contactless Vital Signs and Activity Monitoring of In-Vivo Rodents Using a mm-Wave FMCW Radar
arXiv (Cornell University)·2025Open Access
N-Terminal deleted isoforms of E3 ligase RNF220 are ubiquitously expressed and required for mouse muscle differentiation
Molecules and Cells·2025· 1 citedOpen Access
Telomerase reverse transcriptase degradation via a rationally designed covalent proteolysis targeting chimera
Bioorganic & Medicinal Chemistry Letters·2025
Abstract 1970 Defining roles of telomerase reverse transcriptase in DNA damage response
Journal of Biological Chemistry·2025Open Access
Abstract 639: Targeting TET3 potentiates an effective anti-tumor immune response after irradiation
Cancer Research·2025· 1 cited
Abstract 4199: Defining roles of telomerase reverse transcriptase in DNA damage response
Cancer Research·2025
Abstract 5178: Ascites conditioning reduces latency in vivo for progression of ID8 TP53 KO murine ovarian cancer model
Cancer Research·2025
Abstract 4098: PARP1 and EZH2 independently limit 5'-3' end resection to promote rapid DNA repair after radiation
Cancer Research·2025
Abstract 6877: Oxidative stress induced lipid peroxidation as a mechanism of topoisomerase II alpha poisoning by chemotherapeutic agents
Cancer Research·2025
N-Terminal Deleted Isoforms of E3 Ligase RNF220 (Isoform 4) Are Ubiquitously Expressed and Required for Mouse Muscle Differentiation
2025Open Access
Timing Anti-PD-L1 Checkpoint Blockade Immunotherapy to Enhance Tumor Irradiation
Cancers·2025· 2 citedOpen Access
Noncanonical inhibition of topoisomerase II alpha by oxidative stress metabolites
Redox Biology·2025Open Access
Telomerase Reverse Transcriptase Degradation Via a Rationally Designed Covalent Proteolysis Targeting Chimera
SSRN Electronic Journal·2025Open Access
Could senescent cells be the prescription for therapeutic cancer vaccines?
Immunotherapy·2024· 1 citedOpen Access
Timing anti-PD-L1 checkpoint blockade immunotherapy to enhance tumor irradiation
2024Open Access
Therapy-Induced Cellular Senescence: Potentiating Tumor Elimination or Driving Cancer Resistance and Recurrence?
Cells·2024· 26 citedOpen Access
Context-dependent roles for autophagy in myeloid cells in tumor progression
2024· 1 citedOpen Access
Telomerase Reverse Transcriptase Degradation via a Rationally Designed Covalent Proteolysis Targeting Chimera
2024· 1 citedOpen Access
Telomerase Reverse Transcriptase Degradation via a Rationally Designed Covalent Proteolysis Targeting Chimera
2024· 1 citedOpen Access
Telomerase Reverse Transcriptase Degradation via a Rationally Designed Covalent Proteolysis Targeting Chimera
2024· 1 citedOpen Access
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Frequent Co-authors

James A. Spudich(6), Ralph R. Weichselbaum(5), Donald J. Wolfgeher(5), Ali Javaheri(4), Jacques Côté(4), Yoko Y. Toyoshima(4), John S. Choy(4), Andrew W. Truman(4), J A Spudich(3), Taro Q.P. Uyeda(3), Robert Wysocki(3), Gerald R. Fink(3), Chrisostomos Prodromou(3), Elena V. Efimova(3), Helena J. Mauceri(3), Thomas E. Darga(3), Joon H. Huh(2), Stéphane Allard(2), Olivier Jobin‐Robitaille(2), Michael A. Beckett(2)