Bellina A.S. Mushala

University of Pittsburgh — United States

Biochemistry, Genetics and Molecular Biology · Life Sciences

5h-index104citations27works0.32yr avg
OpenAlex↗ORCID↗Google Scholar↗Lab page↗

Accepting Students?

No reports yet. Know if this professor is taking students?

Research Topics

Mitochondrial Function and Pathology(13), Adipose Tissue and Metabolism(12), Cardiovascular Function and Risk Factors(7), Diet and metabolism studies(7), ATP Synthase and ATPases Research(4)

Publications27 total

Essential Role for Trf2 in Cardiac Development and Function
2025Open Access
<scp>GCN5L1</scp> Inhibits Pyruvate Dehydrogenase Phosphorylation During Cardiac Ischemia–Reperfusion Injury
FASEB BioAdvances·2025Open Access
Adropin protects against cardiac remodeling and metabolic dysfunction in a mouse model of HFpEF
2025Open Access
Cardiac-specific GCN5L1 deficiency promotes MASLD in HFpEF
2025Open Access
Abstract Tu042: Fatty Acid Dysregulation Drives Cardio-Hepatic Crosstalk in Heart Failure with Preserved Ejection Fraction (HFpEF)
Circulation Research·2024
Abstract Mo069: Phospholamban Acetylation Enhances Cardiomyocyte Calcium Cycling Under Conditions of High-Fat Feeding
Circulation Research·2024· 1 cited
TCA Cycle Dysfunction and Amino Acid Catabolism Drive Hepatic Steatosis in Mice with HFpEF
2024Open Access
GCN5L1 inhibits pyruvate dehydrogenase phosphorylation during cardiac ischemia-reperfusion injury
2024Open Access
Adropin Treatment Restores Cardiac Function and Metabolic Flexibility in Diabetic Cardiomyopathy
Physiology·2024
Validation of GCN5L1/BLOC1S1/BLOS1 antibodies using knockout cells and tissue
Biochemical Journal·2024· 2 citedOpen Access
GCN5L1-mediated acetylation prevents Rictor degradation in cardiac cells after hypoxic stress
Cellular Signalling·2024Open Access
Abstract 17305: Diet-Induced Obesity Drives Phospholamban Acetylation and Promotes Cardiomyocyte Calcium Reuptake Into the Sarcoplasmic Reticulum
Circulation·2023
GCN5L1-mediated acetylation prevents Rictor degradation in cardiac cells after hypoxic stress
2023Open Access
Validation of GCN5L1/BLOC1S1/BLOS1 Antibodies Using Knockout Cells and Tissue
2023Open Access
Loss of GPR19 drives obesity-related cardiac dysfunction in mice
Physiology·2023· 1 cited
G-protein coupled receptor 19 (GPR19) knockout mice display sex-dependent metabolic dysfunction
Scientific Reports·2023· 7 citedOpen Access
G-PROTEIN COUPLED RECEPTOR 19 (GPR19) KNOCKOUT MICE DISPLAY SEX-DEPENDENT METABOLIC DYSFUNCTION
2022Open Access
Abstract P3050: The Adropin-GPR19 Signaling Axis In Diabetic Cardiomyopathy
Circulation Research·2022
<scp>GCN5L1</scp> impairs diastolic function in mice exposed to a high fat diet by restricting cardiac pyruvate oxidation
Physiological Reports·2022· 24 citedOpen Access
BS3 GCN5L1 promotes diastolic dysfunction by inhibiting cardiac pyruvate oxidation
2022· 1 citedOpen Access
page 1 of 2Next →

Frequent Co-authors

Iain Scott(25), Michael W. Stoner(21), Janet R. Manning(20), Paramesha Bugga(18), Michael J. Jurczak(15), Dharendra Thapa(12), Bingxian Xie(10), Brenda McMahon(8), Amber Vandevender(7), Nisha Bhattarai(7), Maryam Sharifi‐Sanjani(7), Xuemei Zeng(3), Nathan A. Yates(3), Lia R. Edmunds(3), Manling Zhang(3), Ian Sipula(3), John Sembrat(3), Brett A. Kaufman(3), Sruti S. Shiva(3), Stacy L. Gelhaus(3)