Günter Tremp

Medicine · Health Sciences

20h-index2.9kcitations36works0.02yr avg
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Research Topics

Alzheimer's disease research and treatments(18), Prion Diseases and Protein Misfolding(6), Cholinesterase and Neurodegenerative Diseases(5), Nuclear Receptors and Signaling(5), Computational Drug Discovery Methods(4)

Publications36 total

Expression of human FE65 in amyloid precursor protein transgenic mice is associated with a reduction in β‐amyloid load
Journal of Neurochemistry·2005· 50 cited
Akt3 overexpression in the heart results in progression from adaptive to maladaptive hypertrophy
Journal of Molecular and Cellular Cardiology·2005· 104 cited
Massive CA1/2 Neuronal Loss with Intraneuronal and N-Terminal Truncated Aβ42 Accumulation in a Novel Alzheimer Transgenic Model
American Journal Of Pathology·2004· 399 cited
Robust functional gene validation by adenoviral vectors: one-step Escherichia coli-Derived Recombinant Adenoviral Genome construction
Gene Therapy·2004· 10 cited
S1-02-01 Amino-truncated abeta 42 species as early diagnostic and etiological biomarkers of Alzheimer's disease
Neurobiology of Aging·2004· 1 cited
P2-077 Expression of human FE65 in APP transgenic mice is associated with a reduction in β-amyloid load
Neurobiology of Aging·2004
Hippocampal Neuron Loss Exceeds Amyloid Plaque Load in a Transgenic Mouse Model of Alzheimer's Disease
American Journal Of Pathology·2004· 280 citedOpen Access
Hippocampal neuron loss is greater than amyloid plaque load in a transgenic mouse model of Alzheimer’s disease
Open Research Online (The Open University)·2004
Characterisation of cytoskeletal abnormalities in mice transgenic for wild-type human tau and familial Alzheimer's disease mutants of APP and presenilin-1
Neurobiology of Disease·2003· 88 citedOpen Access
Time sequence of maturation of dystrophic neurites associated with Aβ deposits in APP/PS1 transgenic mice
Experimental Neurology·2003· 279 cited
Neurons overexpressing mutant presenilin‐1 are more sensitive to apoptosis induced by endoplasmic reticulum‐Golgi stress
Journal of Neuroscience Research·2002· 70 cited
Intraneuronal APP/Aβ Trafficking and Plaque Formation in β‐Amyloid Precursor Protein and Presenilin‐1 Transgenic Mice
Brain Pathology·2002· 126 citedOpen Access
Increased tau phosphorylation but absence of formation of neurofibrillary tangles in mice double transgenic for human tau and Alzheimer mutant (M146L) presenilin-1
Neuroscience Letters·2002· 40 cited
Reelin in plaques of β-amyloid precursor protein and presenilin-1 double-transgenic mice
Neuroscience Letters·2001· 59 cited
Intraneuronal Aβ accumulation precedes plaque formation in β-amyloid precursor protein and presenilin-1 double-transgenic mice
Neuroscience Letters·2001· 336 cited
Neurofibrillary tangles and tau phosphorylation
Biochemical Society Symposia·2001· 137 cited
Expression of amyloid precursor protein and presenilin-1 in transgenic mice
Neurobiology of Aging·2000· 1 cited
Progression of neuropathology in transgenic mice expressing genes linked to familial Alzheimer's disease: APP, PS-1 or both
Neurobiology of Aging·2000
Presenilins and Alzheimer’s disease: biological functions and pathogenic mechanisms
Progress in Neurobiology·2000· 155 cited
Transgenic Expression of the Shortest Human Tau Affects Its Compartmentalization and Its Phosphorylation as in the Pretangle Stage of Alzheimer's Disease
American Journal Of Pathology·1999· 191 cited
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Frequent Co-authors

Laurent Pradier(20), Véronique Blanchard(14), Christian Czech(13), Nathalie Touchet(10), Oliver Wirths(7), Thomas A. Bayer(7), Saliha Moussaoui(6), Bruno Bonici(5), Gerd Multhaup(5), Brigitte Schombert(5), Luc Mercken(5), Nicolas Sergeant(4), Konrad Beyreuther(4), Jean‐Pierre Brion(4), Didier Boquet(4), E. Hecker(4), André Delacourte(3), Christoph Schmitz(3), Dominique Langui(3), Charles Duyckaerts(3)