Hideki Enomoto

Kobe University — Japan

Medicine · Health Sciences

43h-index9.7kcitations131works1.02yr avg
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Research Topics

Congenital gastrointestinal and neural anomalies(42), Intestinal Malrotation and Obstruction Disorders(24), Neurogenesis and neuroplasticity mechanisms(15), Gastrointestinal motility and disorders(13), Neuroblastoma Research and Treatments(13)

Publications101 total

A <i>Gfrα3</i> Conditional Knockout Mouse Reveals a Requirement for <scp>GFRα3</scp> / <scp>RET</scp> Signaling in Cholecystokinin‐Positive Enteroendocrine Cells
genesis·2026Open Access
Intramuscular enteric glia persist in Hirschsprung disease and undergo neurogenesis in response to GDNF-NCAM1 signaling
Scientific Reports·2025· 1 citedOpen Access
The long‐term survival of enteroendocrine cells depends on their subtype and is linked to peripheral sensory innervation
Development Growth & Differentiation·2025· 2 citedOpen Access
Pcgf1 gene disruption reveals primary involvement of epigenetic mechanism in neuronal subtype specification in the enteric nervous system
Development Growth & Differentiation·2023· 4 citedOpen Access
A Single RET Mutation in Hirschsprung Disease Induces Intestinal Aganglionosis Via a Dominant-Negative Mechanism
Cellular and Molecular Gastroenterology and Hepatology·2022· 9 citedOpen Access
Uts2b is a microbiota-regulated gene expressed in vagal afferent neurons connected to enteroendocrine cells producing cholecystokinin
Biochemical and Biophysical Research Communications·2022· 11 citedOpen Access
Live visualization of a functional RET‐EGFP chimeric receptor in homozygous knock‐in mice
Development Growth & Differentiation·2021· 4 citedOpen Access
Enhanced enteric neurogenesis by Schwann cell precursors in mouse models of Hirschsprung disease
Glia·2021· 32 cited
Increased RET Activity Coupled with a Reduction in the<i>RET</i>Gene Dosage Causes Intestinal Aganglionosis in Mice
eNeuro·2021· 6 citedOpen Access
Point mutagenesis in mouse reveals contrasting pathogenetic effects between MEN2B‐ and Hirschsprung disease‐associated missense mutations of the <i>RET</i> gene
Development Growth & Differentiation·2020· 11 citedOpen Access
Generation of conditional <i>ALK F1174L</i> mutant mouse models for the study of neuroblastoma pathogenesis
genesis·2019· 14 cited
Mice conditionally expressing RET(C618F) mutation display C cell hyperplasia and hyperganglionosis of the enteric nervous system
genesis·2019· 6 citedOpen Access
Technologies for Live Imaging of Enteric Neural Crest-Derived Cells
Methods in molecular biology·2019· 2 cited
Contributors
Elsevier eBooks·2018
Development of the Enteric Nervous System
Elsevier eBooks·2018· 1 cited
Catenins Steer Cell Migration via Stabilization of Front-Rear Polarity
Developmental Cell·2017· 42 citedOpen Access
Dual origin of enteric neurons in vagal Schwann cell precursors and the sympathetic neural crest
Proceedings of the National Academy of Sciences·2017· 132 citedOpen Access
FANTOM5 CAGE profiles of human and mouse samples
Scientific Data·2017· 350 citedOpen Access
Striatal hypodopamine phenotypes found in transgenic mice that overexpress glial cell line-derived neurotrophic factor
Neuroscience Letters·2017· 6 cited
Retrograde transport of neurotrophic factor signaling: implications in neuronal development and pathogenesis
The Journal of Biochemistry·2016· 72 cited
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Frequent Co-authors

Jeffrey Milbrandt(16), Heather M. Young(10), Toshihiro Uesaka(10), Eugene M. Johnson(7), Robert O. Heuckeroth(7), Mayumi Nagashimada(6), Peter Arner(5), Judith P. Golden(4), Annette J. Bergner(4), Richard B. Anderson(4), Donald F. Newgreen(4), Shigenobu Yonemura(4), Alistair R. R. Forrest(3), Hideya Kawaji(3), J. Kenneth Baillie(3), Timo Lassmann(3), Masayoshi Itoh(3), Yuri Ishizu(3), Magda Babina(3), Anthony G Beckhouse(3)