Jane Tulloch

Discovery Centre — Canada

Medicine · Health Sciences

15h-index1.5kcitations44works4.92yr avg
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Research Topics

Alzheimer's disease research and treatments(33), Neuroscience and Neuropharmacology Research(21), Neuroinflammation and Neurodegeneration Mechanisms(15), Computational Drug Discovery Methods(6), Cholinesterase and Neurodegenerative Diseases(5)

Publications44 total

Amyloid‐Beta Pathology Increases Synaptic Engulfment by Glia in Feline Cognitive Dysfunction Syndrome: A Naturally Occurring Model of Alzheimer's Disease
European Journal of Neuroscience·2025· 3 citedOpen Access
Evidence for trans-synaptic propagation of oligomeric tau in human progressive supranuclear palsy
Nature Neuroscience·2025· 12 citedOpen Access
Author response for "Amyloid-Beta Pathology Increases Synaptic Engulfment by Glia in Feline Cognitive Dysfunction Syndrome: A Naturally Occurring Model of Alzheimer's Disease"
2025
Author response for "Amyloid-Beta Pathology Increases Synaptic Engulfment by Glia in Feline Cognitive Dysfunction Syndrome: A Naturally Occurring Model of Alzheimer's Disease"
2025
Divergent actions of physiological and pathological amyloid-β on synapses in live human brain slice cultures
Nature Communications·2025· 10 citedOpen Access
Amyloid-beta pathology increases synaptic engulfment by glia in feline cognitive dysfunction syndrome: A naturally occurring model of Alzheimer’s disease
2025Open Access
Microglia determine ß-amyloid plaque burden but are non-essential for downstream pathology
2024Open Access
Author response for "Synaptic gene expression changes in frontotemporal dementia due to the MAPT 10+16 mutation"
2024
Synaptic gene expression changes in frontotemporal dementia due to the <i>MAPT</i> 10 + 16 mutation
Neuropathology and Applied Neurobiology·2024· 4 citedOpen Access
Author response for "Synaptic gene expression changes in frontotemporal dementia due to the MAPT 10+16 mutation"
2024
Synaptic gene expression changes in frontotemporal dementia due to the <i>MAPT</i> 10+16 mutation
2024· 5 citedOpen Access
Opposing roles of physiological and pathological amyloid-β on synapses in live human brain slice cultures
2024· 3 citedOpen Access
Transmembrane protein 97 is a potential synaptic amyloid beta receptor in human Alzheimer’s disease
Acta Neuropathologica·2024· 27 citedOpen Access
Human astrocytes and microglia show augmented ingestion of synapses in Alzheimer’s disease via MFG-E8
Cell Reports Medicine·2023· 76 citedOpen Access
Synaptic oligomeric tau in Alzheimer’s disease — A potential culprit in the spread of tau pathology through the brain
Neuron·2023· 182 citedOpen Access
Synaptic oligomeric tau in Alzheimer’s disease – a potential culprit in the spread of tau pathology through the brain
2023· 20 citedOpen Access
Apolipoprotein E isoform does not influence trans-synaptic spread of tau pathology in a mouse model
Brain and Neuroscience Advances·2023· 11 citedOpen Access
Synaptic resilience is associated with maintained cognition during ageing
Alzheimer s & Dementia·2022· 31 citedOpen Access
Evidence for trans-synaptic propagation of oligomeric tau in Progressive Supranuclear Palsy
2022· 8 citedOpen Access
Synaptic resilience is associated with maintained cognition during ageing
2022· 5 citedOpen Access
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Frequent Co-authors

Tara L. Spires‐Jones(41), Jamie Rose(31), Colin Smith(29), Makis Tzioras(24), Declan King(22), Martí Colom‐Cadena(17), Robert I. McGeachan(15), Christopher M. Henstridge(15), C. T. M. Davies(14), Owen Dando(13), Rosemary J. Jackson(13), Abigail G. Herrmann(12), Giles E. Hardingham(11), Chris-Anne McKenzie(9), Monique Hooley(8), Jamie McQueen(8), Xin He(8), Kristján Holt(8), Douglas J. Lamont(8), Paul Baxter(7)