Anne Landsem

Nordland Hospital Trust — Norway

Medicine · Health Sciences

11h-index277citations31works0.02yr avg
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Research Topics

Blood Coagulation and Thrombosis Mechanisms(9), Platelet Disorders and Treatments(7), Complement system in diseases(7), Hemophilia Treatment and Research(6), Coagulation, Bradykinin, Polyphosphates, and Angioedema(6)

Publications31 total

Impact of Preanalytical Storage Time and Air Exposure on ROTEM and Coagulation Sample Stability
Advances in Hematology·2026Open Access
138 Escherichia coli-induced platelet aggregation in human whole blood is partly dependent on complement C3b
Immunobiology·2023
Complement C3b contributes to Escherichia coli-induced platelet aggregation in human whole blood
Frontiers in Immunology·2022· 11 citedOpen Access
Dental and Periodontal Health in Acute Intermittent Porphyria
Life·2022· 5 citedOpen Access
Venous Air Embolism Activates Complement C3 Without Corresponding C5 Activation and Trigger Thromboinflammation in Pigs
Frontiers in Immunology·2022· 11 citedOpen Access
A Conformational Change of Complement C5 Is Required for Thrombin-Mediated Cleavage, Revealed by a Novel Ex Vivo Human Whole Blood Model Preserving Full Thrombin Activity
The Journal of Immunology·2021· 19 citedOpen Access
Cholesterol Crystals Induce Coagulation Activation through Complement-Dependent Expression of Monocytic Tissue Factor
The Journal of Immunology·2019· 38 citedOpen Access
The role of complement and Toll-like receptors in thromboinflammation
Duo Research Archive (University of Oslo)·2019Open Access
Phagocytosis of live and dead Escherichia coli and Staphylococcus aureus in human whole blood is markedly reduced by combined inhibition of C5aR1 and CD14
Molecular Immunology·2019· 11 citedOpen Access
Lifestyle factors including diet and biochemical biomarkers in acute intermittent porphyria: Results from a case-control study in northern Norway
Molecular Genetics and Metabolism·2018· 26 citedOpen Access
Complement component 5 does not interfere with physiological hemostasis but is essential for<i>Escherichia coli-</i>induced coagulation accompanied by Toll-like receptor 4
Clinical & Experimental Immunology·2018· 8 citedOpen Access
A novel human whole blood model preventing fibrin formation reveals that thrombin does not cleave plasma C5 under physiological conditions
Molecular Immunology·2018
Combined C5aR1 and CD14 inhibition significantly reduced cytokine release induced by both viable and heat inactivated bacteria in human whole blood
Molecular Immunology·2017
Complement C5, phagocytosis and Toll-like receptor 4 play key roles in Escherichia coli- induced surface expression of tissue factor on human monocytes
Molecular Immunology·2017
Complement and coagulation is activated in periodontitis in acute intermittent porphyria
Molecular Immunology·2017
Characterization of a novel whole blood model for the study of thrombin in complement activation and inflammation
Molecular Immunology·2017· 1 cited
A complement-dependent human long-noncoding RNA induced by Staphylococcus aureus
Molecular Immunology·2017
Systemic inflammation in acute intermittent porphyria: a case–control study
Clinical & Experimental Immunology·2016· 29 citedOpen Access
The effect of combined inhibition of C5aR1 and CD14 on phagocytosis of E. coli in human whole blood
Immunobiology·2016
Complement C5 is essential for Escherichia coli-induced coagulation activation, independent of inhibition of cytokines inducing tissue factor
Immunobiology·2016
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Frequent Co-authors

Tom Eirik Mollnes(25), Hilde Fure(21), Ole‐Lars Brekke(18), Dorte Christiansen(16), Erik Waage Nielsen(12), Judith Krey Ludviksen(11), Terje Espevik(7), John D. Lambris(6), Bård Ove Karlsen(6), Grethe Bergseth(6), Trent M. Woodruff(5), Per H. Nilsson(5), Elin Storjord(4), Jim André Dahl(4), Corinna Lau(4), Monica Mathisen(4), O-L Brekke(3), Knut Tore Lappegård(3), Stig Nymo(3), Søren Erik Pischke(3)