E. T. Sutton

Medicine · Health Sciences

14h-index582citations43works0.02yr avg
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Research Topics

Alzheimer's disease research and treatments(7), Nitric Oxide and Endothelin Effects(6), Eicosanoids and Hypertension Pharmacology(5), Neurological Disease Mechanisms and Treatments(5), Inflammatory mediators and NSAID effects(4)

Publications43 total

Trends in floating kelp along the Strait of Juan de Fuca and the outer coast (WA, USA): patterns and links to environmental drivers
Western CEDAR (Western Washington University)·2017
Indigo Carmine-Induced Hypotension in a Parturient With Idiopathic Pulmonary Artery Hypertension, Hypertrophic Cardiomyopathy, and LAD Myocardial Bridging
Journal of Cardiothoracic and Vascular Anesthesia·2015· 5 cited
Aging enhances vascular dysfunction induced by the Alzheimer's peptide β-amyloid
Neurological Research·2004· 36 cited
Development of CD30+ lymphoproliferative disease in mice lacking interferon regulatory factor-1
Oncogene·2003· 3 citedOpen Access
The Therapeutic Effects of Epidural Intercellular Adhesion Molecule-1 Monoclonal Antibody in a Rabbit Model: Involvement of the Intercellular Adhesion Molecule-1 Pathway in Spinal Cord Ischemia
Anesthesia & Analgesia·2003· 9 cited
Physiological levels of<i>β</i>-amyloid induce cerebral vessel dysfunction and reduce endothelial nitric oxide production
Neurological Research·2001· 80 cited
Potassium channel openers prevent β-amyloid toxicity in bovine vascular endothelial cells
Neuroscience Letters·2000· 27 cited
Animal Model of Alzheimer‐like Vascular Pathology and Inflammatory Reaction
Annals of the New York Academy of Sciences·2000· 14 cited
Estrogen protects peripheral and cerebral blood vessels from toxicity of Alzheimer peptide amyloid-beta and inflammatory reaction.
PubMed·1999· 27 cited
Animal model of vascular inflammation.
PubMed·1999· 11 cited
Amyloid-beta peptide induced inflammatory reaction is mediated by the cytokines tumor necrosis factor and interleukin-1.
PubMed·1999· 46 cited
The protective effect of K+ channel openers on ^-amyloid induced cerebrovascular endothelial dysfunction
Neurological Research·1999· 17 cited
EPIDURAL ICAM-1 MONOCLONAL ANTIBODY DECREASES LEUKOCYTE RECRUITMENT AND INFILTRATION AND OFFERS NEUROPROTECTION AFTER SPINAL CORD ISCHEMIA
Journal of Neurosurgical Anesthesiology·1998
EPIDURAL ICAM-1 MONOCLONAL ANTIBODY DECREASES LEUKOCYTE RECRUITMENT AND INFILTRATION AND OFFERS NEUROPROTECTION AFTER SPINAL CORD ISCHEMIA
Anesthesiology·1998
Evidence for leukocyte infiltration, ICAM-1 expression, permeability following ischemic paraplegia: Functional blocking of ICAM-1 as a new therapeutic target
The FASEB Journal·1998
β-Amyloid-Induced Coronary Artery Vasoactivity and Endothelial Damage
Journal of Cardiovascular Pharmacology·1997· 18 cited
β‐Amyloid‐Induced Cerebrovascular Endothelial Dysfunction
Annals of the New York Academy of Sciences·1997· 30 cited
In vivo vascular damage, leukocyte activation and inflammatory response induced by beta-amyloid.
PubMed·1997· 21 cited
Cerebrovascular endothelial dysfunction mediated by β-amyloid
Neuroreport·1997· 58 cited
ENDOTHELIAL STRUCTURAL INTEGRITY IS MAINTAINED DURING ENDOTOXIC SHOCK IN AN INTERLEUKIN-1 TYPE 1 RECEPTOR KNOCKOUT MOUSE
Shock·1997· 16 cited
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Frequent Co-authors

C. H. Baker(16), J. M. Price(10), Tom Thomas(5), M. Bryant(5), Johannes A.G. Rhodin(5), D. L. Davis(5), Gary Hellermann(4), Tom Thomas(4), Xuedong Chi(3), Eugene S. Fu(3), Leon D. Prockop(3), Donald L. Davis(3), Chris McLendon(2), Alison Hellermann(2), John R. Dietz(2), Kamatham A. Naidu(2), C. H. Baker(2), F. R. Wilmoth(2), Bruce G. Lindsey(2), Carleton H. Baker(2)