Natalia Podlutskaya

The University of Texas Health Science Center at San Antonio — United States

Biochemistry, Genetics and Molecular Biology · Life Sciences

8h-index1.7kcitations15works0.02yr avg
OpenAlex↗Google Scholar↗Lab page↗

Accepting Students?

No reports yet. Know if this professor is taking students?

Research Topics

Genetics, Aging, and Longevity in Model Organisms(10), Mitochondrial Function and Pathology(5), Alzheimer's disease research and treatments(4), Circadian rhythm and melatonin(3), Adipose Tissue and Metabolism(3)

Publications15 total

Corrigendum to “Chronic inhibition of mTOR by rapamycin modulates cognitive and non-cognitive components of behavior throughout lifespan in mice” [Neuroscience 223 (2012) 102–113]
Neuroscience·2015Open Access
Decreased <i>in vitro</i> Mitochondrial Function is Associated with Enhanced Brain Metabolism, Blood Flow, and Memory in Surfl-Deficient Mice
Journal of Cerebral Blood Flow & Metabolism·2013· 35 citedOpen Access
Over‐expression of heat shock factor 1 phenocopies the effect of chronic inhibition of <scp>TOR</scp> by rapamycin and is sufficient to ameliorate Alzheimer's‐like deficits in mice modeling the disease
Journal of Neurochemistry·2012· 81 citedOpen Access
Environmental enrichment prevents astroglial pathological changes in the hippocampus of APP transgenic mice, model of Alzheimer's disease
Experimental Neurology·2012· 174 cited
Chronic inhibition of mammalian target of rapamycin by rapamycin modulates cognitive and non-cognitive components of behavior throughout lifespan in mice
Neuroscience·2012· 213 citedOpen Access
Testing the Oxidative Stress Hypothesis of Aging in Primate Fibroblasts: Is There a Correlation Between Species Longevity and Cellular ROS Production?
The Journals of Gerontology Series A·2012· 60 citedOpen Access
Mouse Health Span: Why Lifespan is No Longer Enough
Europe PMC (PubMed Central)·2012
Correction: Inhibition of mTOR by Rapamycin Abolishes Cognitive Deficits and Reduces Amyloid-β Levels in a Mouse Model of Alzheimer's Disease
PLoS ONE·2011· 2 citedOpen Access
Correction: Inhibition of mTOR by Rapamycin Abolishes Cognitive Deficits and Reduces Amyloid-β Levels in a Mouse Model of Alzheimer's Disease
PLoS ONE·2011· 2 citedOpen Access
P3‐151: mTOR regulates chaperone networks and cognitive outcomes in mice modeling Alzheimer's disease
Alzheimer s & Dementia·2011
Inflammation in Aging and Age-related disease, San Antonio Nathan Shock Aging Center 2011 Conference on Aging, Mayan Ranch, Texas Hill Country, Bandera, TX, USA
Europe PMC (PubMed Central)·2011
Inhibition of mTOR by Rapamycin Abolishes Cognitive Deficits and Reduces Amyloid-β Levels in a Mouse Model of Alzheimer's Disease
PLoS ONE·2010· 993 citedOpen Access
The long lifespan of two bat species is correlated with resistance to protein oxidation and enhanced protein homeostasis
The FASEB Journal·2009· 115 citedOpen Access
Testing hypotheses of aging in long-lived mice of the genus Peromyscus: association between longevity and mitochondrial stress resistance, ROS detoxification pathways, and DNA repair efficiency
AGE·2008· 55 citedOpen Access
Comparative analysis of DNA repair pathways in mammals.
The FASEB Journal·2008· 2 cited

Frequent Co-authors

Verónica Galván(9), Arlan Richardson(7), Randy Strong(7), Steven N. Austad(7), Matthew J. Hart(6), Jonathan J. Halloran(6), Raquel R Burbank(6), Stacy A. Hussong(5), Andrej Podlutsky(4), Patricia Spilman(3), Jayanta Debnath(3), Olivia Gorostiza(3), Dale E. Bredesen(3), Lauren Sloane(3), Adam B. Salmon(3), Anson Pierce(3), Anna Csiszár(3), Zoltán Ungvári(3), Daniel Pulliam(3), K.E. Fischer(2)