Leonard Girnita

Karolinska University Hospital — Sweden

Biochemistry, Genetics and Molecular Biology · Life Sciences

40h-index4.7kcitations181works0.52yr avg
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Research Topics

Receptor Mechanisms and Signaling(51), Estrogen and related hormone effects(33), Cancer, Hypoxia, and Metabolism(32), Growth Hormone and Insulin-like Growth Factors(30), Cancer, Stress, Anesthesia, and Immune Response(28)

Publications181 total

G Protein-coupled and Membrane Tyrosine Kinase Receptor Relationships Yield Therapeutic Opportunities
Endocrine Reviews·2025· 2 cited
Data from Competing engagement of β-arrestin isoforms balances IGF1R/p53 signaling and controls melanoma cell chemotherapeutic responsiveness
2024Open Access
Supplementary Figure 5 from Competing engagement of β-arrestin isoforms balances IGF1R/p53 signaling and controls melanoma cell chemotherapeutic responsiveness
2024Open Access
Supplementary Figure 6 from Competing engagement of β-arrestin isoforms balances IGF1R/p53 signaling and controls melanoma cell chemotherapeutic responsiveness
2024Open Access
Supplementary Figure 6 from Competing engagement of β-arrestin isoforms balances IGF1R/p53 signaling and controls melanoma cell chemotherapeutic responsiveness
2024Open Access
Supplementary Figure 3 from Competing engagement of β-arrestin isoforms balances IGF1R/p53 signaling and controls melanoma cell chemotherapeutic responsiveness
2024Open Access
Table S2 from Competing engagement of β-arrestin isoforms balances IGF1R/p53 signaling and controls melanoma cell chemotherapeutic responsiveness
2024Open Access
Table S1 from Competing engagement of β-arrestin isoforms balances IGF1R/p53 signaling and controls melanoma cell chemotherapeutic responsiveness
2024Open Access
Supplementary Figure 1 from Competing engagement of β-arrestin isoforms balances IGF1R/p53 signaling and controls melanoma cell chemotherapeutic responsiveness
2024Open Access
Supplementary Figure 4 from Competing engagement of β-arrestin isoforms balances IGF1R/p53 signaling and controls melanoma cell chemotherapeutic responsiveness
2024Open Access
Supplementary Figure 7 from Competing engagement of β-arrestin isoforms balances IGF1R/p53 signaling and controls melanoma cell chemotherapeutic responsiveness
2024Open Access
Supplementary Figure 2 from Competing engagement of β-arrestin isoforms balances IGF1R/p53 signaling and controls melanoma cell chemotherapeutic responsiveness
2024Open Access
Supplementary Figure 8 from Competing engagement of β-arrestin isoforms balances IGF1R/p53 signaling and controls melanoma cell chemotherapeutic responsiveness
2024Open Access
Supplementary Figure 2 from Competing engagement of β-arrestin isoforms balances IGF1R/p53 signaling and controls melanoma cell chemotherapeutic responsiveness
2024Open Access
Supplementary Figure 3 from Competing engagement of β-arrestin isoforms balances IGF1R/p53 signaling and controls melanoma cell chemotherapeutic responsiveness
2024Open Access
Table S2 from Competing engagement of β-arrestin isoforms balances IGF1R/p53 signaling and controls melanoma cell chemotherapeutic responsiveness
2024Open Access
Supplementary Figure 1 from Competing engagement of β-arrestin isoforms balances IGF1R/p53 signaling and controls melanoma cell chemotherapeutic responsiveness
2024Open Access
Supplementary Figure 8 from Competing engagement of β-arrestin isoforms balances IGF1R/p53 signaling and controls melanoma cell chemotherapeutic responsiveness
2024Open Access
Supplementary Figure 7 from Competing engagement of β-arrestin isoforms balances IGF1R/p53 signaling and controls melanoma cell chemotherapeutic responsiveness
2024Open Access
Supplementary Figure 5 from Competing engagement of β-arrestin isoforms balances IGF1R/p53 signaling and controls melanoma cell chemotherapeutic responsiveness
2024Open Access
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Frequent Co-authors

Olle Larsson(23), Ada Girnita(13), Stefan Seregard(13), Ada Girnita(11), Magnus Axelson(9), Charlotta All‐Ericsson(8), Radu Vasilcanu(7), Armando Bartolazzi(6), Bita Sehat(6), Daiana Vasilcanu(6), Mario A. Economou(6), Caitrin Crudden(6), Claire Worrall(5), Eline Menu(4), Sandra Andersson(4), Huiyuan Zheng(4), Johan Wejde(4), Shin‐Ichiro Takahashi(3), George A. Calin(3), Baoqing Chen(3)