Thomas Söllner

Heidelberg University — Germany

Biochemistry, Genetics and Molecular Biology · Life Sciences

52h-index16.8kcitations114works4.22yr avg
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Research Topics

Cellular transport and secretion(77), Lipid Membrane Structure and Behavior(60), Endoplasmic Reticulum Stress and Disease(18), Mitochondrial Function and Pathology(15), Retinal Development and Disorders(12)

Publications114 total

Mutations of Single Residues in the Complexin N-terminus Exhibit Distinct Phenotypes in Synaptic Vesicle Fusion
Journal of Neuroscience·2024Open Access
Reduced Protein Stability of 11 Pathogenic Missense STXBP1/MUNC18-1 Variants and Improved Disease Prediction
Biological Psychiatry·2024· 5 citedOpen Access
Author Response: SNAP25 disease mutations change the energy landscape for synaptic exocytosis due to aberrant SNARE interactions
2024Open Access
SNAP25 disease mutations change the energy landscape for synaptic exocytosis due to aberrant SNARE interactions
eLife·2024· 16 citedOpen Access
Author Response: SNAP25 disease mutations change the energy landscape for synaptic exocytosis due to aberrant SNARE interactions
2024Open Access
A short perinuclear amphipathic α-helix in Apq12 promotes nuclear pore complex biogenesis
Open Biology·2021· 16 citedOpen Access
Author response for "A short perinuclear amphipathic α-helix in Apq12 promotes nuclear pore complex biogenesis"
2021
A short perinuclear amphipathic α-helix in Apq12 promotes nuclear pore complex biogenesis
2021· 1 citedOpen Access
The Interaction of Munc18-1 Helix 11 and 12 with the Central Region of the VAMP2 SNARE Motif Is Essential for SNARE Templating and Synaptic Transmission
eNeuro·2020· 36 citedOpen Access
Arrangements of proteins at reconstituted synaptic vesicle fusion sites depend on membrane separation
FEBS Letters·2020· 12 citedOpen Access
Complexin Suppresses Spontaneous Exocytosis by Capturing the Membrane-Proximal Regions of VAMP2 and SNAP25
Cell Reports·2020· 50 citedOpen Access
Complexin suppresses spontaneous exocytosis by capturing the membrane-proximal regions of VAMP2 and SNAP25
2019· 6 citedOpen Access
Synaptic vesicle fusion: today and beyond
Nature Structural & Molecular Biology·2019· 27 cited
An Electrostatic Energy Barrier for SNARE-Dependent Spontaneous and Evoked Synaptic Transmission
Cell Reports·2019· 42 citedOpen Access
Uncovering the “secret” lives of vacuolar fusion pores in living cells
The EMBO Journal·2018· 1 citedOpen Access
Tyrosine phosphorylation of Munc18‐1 inhibits synaptic transmission by preventing <scp>SNARE</scp>  assembly
The EMBO Journal·2017· 44 citedOpen Access
Electrostatic anchoring precedes stable membrane attachment of SNAP25/SNAP23 to the plasma membrane
eLife·2017· 28 citedOpen Access
Interactions Between SNAP-25 and Synaptotagmin-1 Are Involved in Vesicle Priming, Clamping Spontaneous and Stimulating Evoked Neurotransmission
Journal of Neuroscience·2016· 80 citedOpen Access
Author response: Electrostatic anchoring precedes stable membrane attachment of SNAP25/SNAP23 to the plasma membrane
2016Open Access
Extension of Helix 12 in Munc18-1 Induces Vesicle Priming
Journal of Neuroscience·2016· 61 citedOpen Access
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Frequent Co-authors

James E. Rothman(22), James A. McNew(12), Francesco Parlati(10), Nikolaus Pfanner(8), Thomas Weber(7), Mariella Ravazzola(6), Walter Neupert(6), James E. Rothman(6), Lelio Orci(5), M Amherdt(5), Jörg Malsam(5), Paul Tempst(4), Alain Perrelet(4), Oleg Varlamov(4), Benedikt Westermann(3), Michael Gmachl(3), Robert J. Johnston(3), Morten Søgaard(3), Rupert Pfaller(3), Walter Nickel(3)