Matthew J. Hangauer

University of California, San Diego — United States

Chemistry · Physical Sciences

18h-index4.3kcitations41works9.92yr avg
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Research Topics

Click Chemistry and Applications(9), Ferroptosis and cancer prognosis(5), RNA modifications and cancer(5), Cancer-related molecular mechanisms research(4), Advanced Fluorescence Microscopy Techniques(4)

Publications41 total

FSP1 and histone deacetylases suppress cancer persister cell ferroptosis
Science Advances·2026Open Access
DNA fragmentation factor B suppresses interferon to enable cancer persister cell regrowth
Nature Cell Biology·2025· 1 citedOpen Access
FSP1 and histone deacetylases suppress cancer persister cell ferroptosis
2025Open Access
DFFB suppresses interferon to enable cancer persister cell regrowth
2025Open Access
Abstract 6792: Understanding and enhancing cancer persister cell ferroptosis sensitivity
Cancer Research·2025
Abstract 1255: Apoptotic DNase DFFB mediates cancer acquired drug resistance by suppressing persister cell intrinsic interferon signaling
Cancer Research·2025
Antigenic cancer persister cells survive direct T cell attack
2025· 2 citedOpen Access
Cancer drug-tolerant persister cells: from biological questions to clinical opportunities
Nature reviews. Cancer·2024· 106 citedOpen Access
Pan-RAF:MEK Molecular Glues Take Center Stage
Cancer Discovery·2024
Abstract 4295: Apoptotic DNase DFFB-induced DNA damage within cancer persister cells underlies acquired resistance to targeted therapies across multiple tumor types
Cancer Research·2024Open Access
Abstract 3964: A cancer survival program against activated T cell attack
Cancer Research·2024
Abstract 99: Apoptotic DNase DFFB mediates cancer persister cell mutagenesis and acquired drug resistance
Cancer Research·2023· 2 cited
Abstract PR003: Apoptotic DNase DFFB mediates stress-induced mutagenesis
Cancer Research·2023
145 An in vitro model of immunotherapy-tolerant melanoma persister cells
Journal of Investigative Dermatology·2019
High-Complexity shRNA Libraries and PI3 Kinase Inhibition in Cancer: High-Fidelity Synthetic Lethality Predictions
Cell Reports·2019· 10 citedOpen Access
Abstract B098: GPX4 is a broadly shared gene vulnerability among residual tumors
Molecular Cancer Therapeutics·2018
Drug-tolerant persister cancer cells are vulnerable to GPX4 inhibition
Nature·2017· 1.6k citedOpen Access
Abstract 1006: Drug-tolerant persister cancer cells are vulnerable to GPX4 inhibition
Cancer Research·2017· 2 cited
Drug Resistance to EGFR Inhibitors in Lung Cancer
Chemotherapy·2016· 99 citedOpen Access
AKT inactivation causes persistent drug tolerance to EGFR inhibitors
Pharmacological Research·2015· 29 cited
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Frequent Co-authors

Carolyn R. Bertozzi(13), Claire E. Turkal(11), David A. Gervasio(10), Anna E. Stuhlfire(10), Brandon E. Mauch(8), Sophie H. Harris(8), Cooper P. Lathrop(8), Michelle H. Paw(8), Michael T. McManus(7), August F. Williams(7), Ariel H. Nguyen(7), Michael Wang(6), Mehrshad Hairani(6), Jennifer L. Page(6), Frank McCormick(5), Vasanthi S. Viswanathan(3), Matthew J. Ryan(3), Dhruv Bole(3), Stuart L. Schreiber(3), Jennifer A. Prescher(3)