Mitchell B. Schaffler

City College of New York — United States

Medicine · Health Sciences

76h-index19.7kcitations188works5.52yr avg
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Research Topics

Bone health and osteoporosis research(74), Bone Metabolism and Diseases(44), Bone fractures and treatments(28), Bone health and treatments(21), Orthopaedic implants and arthroplasty(19)

Publications187 total

The impact of inactivation of the GH/IGF axis during aging on healthspan
GeroScience·2024· 3 citedOpen Access
Targeting mitochondrial dysfunction using methylene blue or mitoquinone to improve skeletal aging
Aging·2024· 8 citedOpen Access
Targeting mitochondrial dysfunction using methylene blue or mitoquinone to improve skeletal aging
2023Open Access
A Fluorescent Intravital Imaging Approach to Study Load-Induced Calcium Signaling Dynamics in Mouse Osteocytes
Journal of Visualized Experiments·2023· 6 cited
Author response for "Lifelong excess in GH elicits sexual dimorphic effects on skeletal morphology and bone mechanical properties"
2022
Author response for "Lifelong excess in GH elicits sexual dimorphic effects on skeletal morphology and bone mechanical properties"
2022
Induction of somatopause in adult mice compromises bone morphology and exacerbates bone loss during aging
Aging Cell·2021· 10 citedOpen Access
Estrogen depletion on In vivo osteocyte calcium signaling responses to mechanical loading
Bone·2021· 28 citedOpen Access
Reply to: A Bisphosphonate With a Low Hydroxyapatite Binding Affinity Prevents Bone Loss in Mice After Ovariectomy and Reverses Rapidly With Treatment Cessation
JBMR Plus·2021· 1 citedOpen Access
Skeletal Response to Insulin in the Naturally Occurring Type 1 Diabetes Mellitus Mouse Model
JBMR Plus·2021· 22 citedOpen Access
A Bisphosphonate With a Low Hydroxyapatite Binding Affinity Prevents Bone Loss in Mice After Ovariectomy and Reverses Rapidly With Treatment Cessation
JBMR Plus·2021· 14 citedOpen Access
Lifelong Excess in GH Elicits Sexually Dimorphic Effects on Skeletal Morphology and Bone Mechanical Properties
Journal of Bone and Mineral Research·2020· 1 citedOpen Access
Apoptotic Osteocytes Induce RANKL Production in Bystanders via Purinergic Signaling and Activation of Pannexin Channels
Journal of Bone and Mineral Research·2020· 40 citedOpen Access
L-Glutamine Increases IGF-1 Liver Expression to Prevent Bone Loss in Sickle Mice
Blood·2019· 2 cited
Issue Information
Annals of the New York Academy of Sciences·2019Open Access
CITED2 mediates the cross‐talk between mechanical loading and IL‐4 to promote chondroprotection
Annals of the New York Academy of Sciences·2019· 28 cited
Correction to: Green tea polyphenol treatment is chondroprotective, anti-inflammatory and palliative in a mouse posttraumatic osteoarthritis model
Arthritis Research & Therapy·2019· 24 citedOpen Access
Mitochondrial Function Is Compromised in Cortical Bone Osteocytes of Long-Lived Growth Hormone Receptor Null Mice
Journal of Bone and Mineral Research·2018· 42 citedOpen Access
Role of pannexin 1 channels in load‐induced skeletal response
Annals of the New York Academy of Sciences·2018· 20 cited
Potential role for a specialized β<sub>3</sub> integrin‐based structure on osteocyte processes in bone mechanosensation
Journal of Orthopaedic Research®·2017· 69 citedOpen Access
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Frequent Co-authors

Robert J. Majeska(15), David P. Fyhrie(10), David B. Burr(7), Sheldon Weinbaum(7), Damien M. Laudier(6), Oran D. Kennedy(5), Karl J. Jepsen(5), Olivier Verborgt(4), Hui Sun(4), Evan L. Flatow(4), Mone Zaidi(3), Deepak Vashishth(3), Stephen C. Cowin(3), Brad C. Herman(3), R. Bruce Martin(2), Eric L. Radin(2), Gary Gibson(2), Kwang‐Wook Choi(2), Yuefeng Han(2), Todd M. Boyce(2)