M. Yamada

Okayama University of Science — Japan

Physics and Astronomy · Physical Sciences

71h-index19.2kcitations429works0.02yr avg
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Research Topics

Particle physics theoretical and experimental studies(135), High-Energy Particle Collisions Research(98), Particle Detector Development and Performance(57), Quantum Chromodynamics and Particle Interactions(46), Neutrino Physics Research(31)

Publications309 total

[Rectosigmoid Colon Cancer with Unresectable Liver Metastases That Responded Remarkably to Bevacizumab-Combined Chemotherapy-A Case Report].
PubMed·2025
A Hidden Danger Behind the Self-Reported Intrauterine Pregnancy
Journal of the American College of Emergency Physicians Open·2025Open Access
Coexistence of IgG4-Related Disease and Renal Anastomosing Hemangioma: A Rare Diagnostic Challenge
Internal Medicine·2025Open Access
Soy protein selectively accumulates formaldehyde
Scientific Reports·2025Open Access
Clinical Significance of Antinuclear Antibodies in Patients with Rheumatoid Arthritis: From SETOUCHI-RA Registry
Journal of Clinical Medicine·2025Open Access
Soy protein selectively accumulates formaldehyde
2024Open Access
[A Case of Submucosal Invasion(SM2)Gastric Cancer with Peritoneal Dissemination Three Years after Surgery].
PubMed·2023
Relationship between imaging parameters and distortion in magnetic resonance images for gamma knife treatment planning
Journal of Applied Clinical Medical Physics·2023· 1 citedOpen Access
The Accumulation of Metal Ions by a Soy Protein–Inorganic Composite Material
Journal of Composites Science·2023· 5 citedOpen Access
Supplementary Table S1-6 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
2023Open Access
Supplementary Figure S1 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
2023Open Access
Supplementary Figure S5 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
2023Open Access
Data from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
2023Open Access
Supplementary Figure S6 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
2023Open Access
Supplementary Figure S2 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
2023Open Access
Supplementary Figure S6 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
2023Open Access
Supplementary Figure S4 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
2023Open Access
Data from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
2023Open Access
Supplementary Figure S5 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
2023Open Access
Supplementary Data from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
2023Open Access
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Frequent Co-authors

Y. Suzuki(25), Keiko Hirata(14), M. Koshiba(14), M. Nakahata(14), Y. Totsuka(14), K. Miyano(14), Y. Oyama(13), T. Tanimori(13), F. M. Newcomer(13), T. Suda(13), T. Kajita(12), N. Sato(12), R. Bayes(12), W. Frati(12), M. Mori(11), H. Miyata(11), H. Takei(11), E.D. Frank(11), R. Van Berg(11), A. K. Mann(10)