M. Mehrdad Tondravi

American Red Cross — United States

Biochemistry, Genetics and Molecular Biology · Life Sciences

17h-index2.8kcitations24works0.02yr avg
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Research Topics

Bone Metabolism and Diseases(10), Bone health and treatments(8), Osteoarthritis Treatment and Mechanisms(5), Cytokine Signaling Pathways and Interactions(5), NF-κB Signaling Pathways(5)

Publications24 total

IL-4 promotes the formation of multinucleated giant cells from macrophage precursors by a STAT6-dependent, homotypic mechanism: contribution of E-cadherin
Journal of Leukocyte Biology·2007· 148 cited
IL-4 suppresses osteoclast development and mature osteoclast function by a STAT6-dependent mechanism: irreversible inhibition of the differentiation program activated by RANKL
Blood·2003· 149 citedOpen Access
NF-κB p50 and p52 Expression Is Not Required for RANK-Expressing Osteoclast Progenitor Formation but Is Essential for RANK- and Cytokine-Mediated Osteoclastogenesis
Journal of Bone and Mineral Research·2002· 160 cited
Regulation of activation-induced receptor activator of NF-κB ligand (RANKL) expression in T cells
European Journal of Immunology·2002
Treatment of a murine model of high-turnover renal osteodystrophy by exogenous BMP-7
Kidney International·2002· 95 citedOpen Access
Regulation of activation-induced receptor activator of NF-κB ligand (RANKL) expression in T cells
European Journal of Immunology·2002· 85 cited
Cortactin potentiates bone metastasis of breast cancer cells.
PubMed·2001· 198 cited
Osteoprotegerin and rank ligand expression in prostate cancer
Urology·2001· 234 cited
Differential Regulation of the Expression of CD95 Ligand, Receptor Activator of Nuclear Factor-κB Ligand (RANKL), TNF-Related Apoptosis-Inducing Ligand (TRAIL), and TNF-α During T Cell Activation
The Journal of Immunology·2001· 34 citedOpen Access
Tumor Necrosis Factor-α (TNF) Stimulates RANKL-induced Osteoclastogenesis via Coupling of TNF Type 1 Receptor and RANK Signaling Pathways
Journal of Biological Chemistry·2001· 488 citedOpen Access
Optimization of Fibroblast Growth Factor-1 as an Anabolic Agent for Osteoporosis
2000
Mice deficient in Abl are osteoporotic and have defects in osteoblast maturation
Nature Genetics·2000· 162 cited
NF-κB and bone: The breaking point
Nature Medicine·1997· 48 cited
Substrate Recognition by Osteoclast Precursors Induces C-src/Microtubule Association
The Journal of Cell Biology·1997· 97 citedOpen Access
Osteoclasts, macrophages, and the molecular mechanisms of bone resorption
Journal of Leukocyte Biology·1997· 113 cited
Osteopetrosis in mice lacking haematopoietic transcription factor PU.1
Nature·1997· 547 cited
OSTEOPETROSIS IN MICE LACKING HEMATOPOIT IC TRANSCRIPTION FACTOR PU. 1
1997· 11 cited
Novel osteoporosis mutation caused by the knock out of the hematopoietic transcription factor PU.1 in transgenic mice
Bone·1996· 5 cited
Cartilage matrix protein forms a type II collagen-independent filamentous network: analysis in primary cell cultures with a retrovirus expression system.
Molecular Biology of the Cell·1995· 63 citedOpen Access
The Role of Coiled-coil α-Helices and Disulfide Bonds in the Assembly and Stabilization of Cartilage Matrix Protein Subunits
Journal of Biological Chemistry·1995· 39 citedOpen Access
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Frequent Co-authors

Steven L. Teitelbaum(6), Paul F. Goetinck(5), José L. Moreno(4), Dominik R. Haudenschild(4), Marisol Quiroz(3), Yousef Abu‐Amer(3), Ruoxiang Wang(3), Yufang Shi(3), Scott R. McKercher(2), Jeanne Erdmann(2), Achsah Keegan(2), F. Patrick Ross(2), Neil Winterbottom(2), Liying Zhang(2), Xiaoren Zhang(2), Christina M. Celluzzi(2), Urs Hofer(2), Kenton L. Anderson(1), R. Maki(1), Yan-Hong Zhang(1)