Pooja Singhmar

The University of Texas MD Anderson Cancer Center — United States

Medicine · Health Sciences

9h-index457citations19works0.02yr avg
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Research Topics

Pain Mechanisms and Treatments(11), Cancer Treatment and Pharmacology(7), Botulinum Toxin and Related Neurological Disorders(4), Receptor Mechanisms and Signaling(3), Epigenetics and DNA Methylation(2)

Publications19 total

Corrigendum to “TTI-101: A competitive inhibitor of STAT3 that spares oxidative phosphorylation and reverses mechanical allodynia in mouse models of neuropathic pain” [Biochem. Pharmacol. 192 (2021) 114688]
Biochemical Pharmacology·2021· 2 citedOpen Access
TTI-101: A competitive inhibitor of STAT3 that spares oxidative phosphorylation and reverses mechanical allodynia in mouse models of neuropathic pain
Biochemical Pharmacology·2021· 41 citedOpen Access
Bexarotene normalizes chemotherapy-induced myelin decompaction and reverses cognitive and sensorimotor deficits in mice
Acta Neuropathologica Communications·2020· 44 citedOpen Access
The fibroblast-derived protein PI16 controls neuropathic pain
Proceedings of the National Academy of Sciences·2020· 72 citedOpen Access
Additional file 2 of Bexarotene normalizes chemotherapy-induced myelin decompaction and reverses cognitive and sensorimotor deficits in mice
Figshare·2020Open Access
Fibroblast-derived PI16 as a novel regulator of neuropathic pain
Figshare·2019Open Access
Fibroblast-derived PI16 as a novel regulator of neuropathic pain
Zenodo (CERN European Organization for Nuclear Research)·2019Open Access
Fibroblast-derived PI16 as a novel regulator of neuropathic pain
Figshare·2019Open Access
PI16 is a non-neuronal regulator of neuropathic pain
2019· 1 citedOpen Access
Orally active Epac inhibitor reverses mechanical allodynia and loss of intraepidermal nerve fibers in a mouse model of chemotherapy-induced peripheral neuropathy
Pain·2018· 49 citedOpen Access
Epac1 interacts with importin β1 and controls neurite outgrowth independently of cAMP and Rap1
Scientific Reports·2016· 17 citedOpen Access
Critical role for Epac1 in inflammatory pain controlled by GRK2-mediated phosphorylation of Epac1
Proceedings of the National Academy of Sciences·2016· 124 citedOpen Access
Critical Role of GRK2 in the Prevention of Chronic Pain
Methods in pharmacology and toxicology·2016
Sensory neuron cAMP signaling in chronic pain
2015· 2 cited
63. GRK2 phosphorylates Epac1 to inhibit signaling and prevent chronic inflammatory pain
Brain Behavior and Immunity·2014
Phosphorylation of Epac1 by GRK2 inhibits Epac1‐Rap1 signaling and prevents chronic pain (802.7)
The FASEB Journal·2014
Angelman Syndrome Protein UBE3A Interacts with Primary Microcephaly Protein ASPM, Localizes to Centrosomes and Regulates Chromosome Segregation
PLoS ONE·2011· 32 citedOpen Access
Mutations in WDR62, encoding a centrosomal and nuclear protein, in Indian primary microcephaly families with cortical malformations
Clinical Genetics·2011· 62 cited
Promoter characterization and regulation of expression of an imprinted gene SLC22A18AS
Gene·2008· 11 cited

Frequent Co-authors

Cobi J. Heijnen(16), Annemieke Kavelaars(13), XiaoJiao Huo(8), Jiacheng Ma(7), Ronnie The Phong Trinh(5), Bo Peng(5), Xiaodong Cheng(4), Niels Eijkelkamp(3), Faiza Baameur(3), Fang Mei(3), Cristina Murga(3), Arun Kumar(3), Annemieke Kavelaars(3), Susana Rojo Berciano(2), David H. Hawke(2), Federico Mayor(2), Angie C.A. Chiang(2), Alexandre V. Seua(2), Luis D. Arroyo(2), Rajasekaran Mahalingam(2)