Liudi Tang

Medicine · Health Sciences

13h-index702citations24works5.32yr avg
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Research Topics

Hepatitis B Virus Studies(12), Hepatitis C virus research(9), RNA modifications and cancer(5), interferon and immune responses(5), Cancer-related molecular mechanisms research(3)

Publications24 total

“PROTAC” modified Dihydroquinolizinones (DHQs) that causes degradation of PAPD-5 and inhibition of hepatitis a virus and hepatitis B virus, in vitro
UNC Libraries·2026Open Access
A hepatitis B virus RNA-sensing and RNA-editing-dependent reporter system
Journal of Virology·2025Open Access
An allosteric inhibitor of sirtuin 2 blocks hepatitis B virus covalently closed circular DNA establishment and its transcriptional activity
Antiviral Research·2024· 8 citedOpen Access
“PROTAC” modified dihydroquinolizinones (DHQs) that cause degradation of PAPD-5 and inhibition of hepatitis A virus and hepatitis B virus, in vitro
Bioorganic & Medicinal Chemistry Letters·2024· 13 citedOpen Access
Mechanism of interferon alpha therapy for chronic hepatitis B and potential approaches to improve its therapeutic efficacy
Antiviral Research·2023· 59 cited
An allosteric inhibitor of sirtuin 2 deacetylase activity exhibits broad-spectrum antiviral activity
Journal of Clinical Investigation·2023· 24 citedOpen Access
Supplementary Tables 1-3, Supplementary Figures 1-5 from Overexpression of RCC2 Enhances Cell Motility and Promotes Tumor Metastasis in Lung Adenocarcinoma by Inducing Epithelial–Mesenchymal Transition
2023Open Access
Data from Overexpression of RCC2 Enhances Cell Motility and Promotes Tumor Metastasis in Lung Adenocarcinoma by Inducing Epithelial–Mesenchymal Transition
2023Open Access
Data from Overexpression of RCC2 Enhances Cell Motility and Promotes Tumor Metastasis in Lung Adenocarcinoma by Inducing Epithelial–Mesenchymal Transition
2023Open Access
Supplementary Tables 1-3, Supplementary Figures 1-5 from Overexpression of RCC2 Enhances Cell Motility and Promotes Tumor Metastasis in Lung Adenocarcinoma by Inducing Epithelial–Mesenchymal Transition
2023Open Access
Heat Shock Protein Family A Member 1 Promotes Intracellular Amplification of Hepatitis B Virus Covalently Closed Circular DNA
Journal of Virology·2022· 3 citedOpen Access
Hepatitis B virus nucleocapsid uncoating: biological consequences and regulation by cellular nucleases
Emerging Microbes & Infections·2021· 30 citedOpen Access
Interferon Alpha Induces Multiple Cellular Proteins That Coordinately Suppress Hepadnaviral Covalently Closed Circular DNA Transcription
Journal of Virology·2020· 31 citedOpen Access
Discovery and Mechanistic Study of a Novel Human-Stimulator-of-Interferon-Genes Agonist
ACS Infectious Diseases·2019· 56 citedOpen Access
DNA Polymerase alpha is essential for intracellular amplification of hepatitis B virus covalently closed circular DNA
PLoS Pathogens·2019· 81 citedOpen Access
Role and mechanism of host cellular DNA repair proteins in hepatitis B virus cccDNA biosynthesis
2019Open Access
Cellular DNA Topoisomerases Are Required for the Synthesis of Hepatitis B Virus Covalently Closed Circular DNA
Journal of Virology·2019· 72 citedOpen Access
Enhancer hijacking: a new direction of research on oncogenesis mechanism
Int J Genet·2018
Virological Basis for the Cure of Chronic Hepatitis B
ACS Infectious Diseases·2018· 56 citedOpen Access
A cell-based high throughput screening assay for the discovery of cGAS-STING pathway agonists
Antiviral Research·2017· 66 citedOpen Access
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Frequent Co-authors

Jinhong Chang(11), Ju‐Tao Guo(11), Junjun Cheng(8), Qiong Zhao(6), Rongwei Guan(5), Jialin Dai(5), Jing Bai(5), Guohua Ji(5), Songbin Fu(5), Nan Wu(5), Muhammad Sheraz(4), Tianlun Zhou(4), Jialei Chen(4), Yanming Du(4), Andrew Snedeker(4), Bo Pang(4), Lin Pang(4), Haiming Sun(4), An Liu(4), Sheng Xiao(4)