Peter Gibbs

The University of Melbourne — Australia

Medicine · Health Sciences

83h-index44.8kcitations1.3kworks1.22yr avg
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Research Topics

Colorectal Cancer Treatments and Studies(407), Genetic factors in colorectal cancer(293), Cancer Genomics and Diagnostics(273), Cancer-related Molecular Pathways(165), Pancreatic and Hepatic Oncology Research(119)

Publications1.3k total

The multiple facets of registry-based randomised controlled trials
Trials·2026Open Access
Figure S9 from The Origin of Highly Elevated Cell-Free DNA in Healthy Individuals and Patients with Pancreatic, Colorectal, Lung, or Ovarian Cancer
2025Open Access
Figure S2 from The Origin of Highly Elevated Cell-Free DNA in Healthy Individuals and Patients with Pancreatic, Colorectal, Lung, or Ovarian Cancer
2025Open Access
Figure S7 from The Origin of Highly Elevated Cell-Free DNA in Healthy Individuals and Patients with Pancreatic, Colorectal, Lung, or Ovarian Cancer
2025Open Access
Supplementary Tables S1 and S2 from The Origin of Highly Elevated Cell-Free DNA in Healthy Individuals and Patients with Pancreatic, Colorectal, Lung, or Ovarian Cancer
2025Open Access
Figure S5 from The Origin of Highly Elevated Cell-Free DNA in Healthy Individuals and Patients with Pancreatic, Colorectal, Lung, or Ovarian Cancer
2025Open Access
Figure S10 from The Origin of Highly Elevated Cell-Free DNA in Healthy Individuals and Patients with Pancreatic, Colorectal, Lung, or Ovarian Cancer
2025Open Access
Figure S4 from The Origin of Highly Elevated Cell-Free DNA in Healthy Individuals and Patients with Pancreatic, Colorectal, Lung, or Ovarian Cancer
2025Open Access
Figure S8 from The Origin of Highly Elevated Cell-Free DNA in Healthy Individuals and Patients with Pancreatic, Colorectal, Lung, or Ovarian Cancer
2025Open Access
Figure S3 from The Origin of Highly Elevated Cell-Free DNA in Healthy Individuals and Patients with Pancreatic, Colorectal, Lung, or Ovarian Cancer
2025Open Access
Figure S1 from The Origin of Highly Elevated Cell-Free DNA in Healthy Individuals and Patients with Pancreatic, Colorectal, Lung, or Ovarian Cancer
2025Open Access
Supplementary Notes 1-5 from The Origin of Highly Elevated Cell-Free DNA in Healthy Individuals and Patients with Pancreatic, Colorectal, Lung, or Ovarian Cancer
2025Open Access
Figure S6 from The Origin of Highly Elevated Cell-Free DNA in Healthy Individuals and Patients with Pancreatic, Colorectal, Lung, or Ovarian Cancer
2025Open Access
Figure S3 from Genotype-Tailored ERK/MAPK Pathway and HDAC Inhibition Rewires the Apoptotic Rheostat to Trigger Colorectal Cancer Cell Death
2025Open Access
Figure S1 from Genotype-Tailored ERK/MAPK Pathway and HDAC Inhibition Rewires the Apoptotic Rheostat to Trigger Colorectal Cancer Cell Death
2025Open Access
Figure S2 from Genotype-Tailored ERK/MAPK Pathway and HDAC Inhibition Rewires the Apoptotic Rheostat to Trigger Colorectal Cancer Cell Death
2025Open Access
Supplementary Table 1 from Genotype-Tailored ERK/MAPK Pathway and HDAC Inhibition Rewires the Apoptotic Rheostat to Trigger Colorectal Cancer Cell Death
2025Open Access
Figure S5 from Genotype-Tailored ERK/MAPK Pathway and HDAC Inhibition Rewires the Apoptotic Rheostat to Trigger Colorectal Cancer Cell Death
2025Open Access
Figure S4 from Genotype-Tailored ERK/MAPK Pathway and HDAC Inhibition Rewires the Apoptotic Rheostat to Trigger Colorectal Cancer Cell Death
2025Open Access
Inequities in mental health services among Aboriginal and Torres Strait Islander people in Australia
Public Health·2025Open Access
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Frequent Co-authors

Jeanne Tie(16), Kenneth W. Kinzler(12), Bert Vogelstein(11), Yuxuan Wang(10), Cristian Tomasetti(10), Joshua D. Cohen(9), Michael Christie(9), Jayesh Desai(9), Oliver M. Sieber(9), Nickolas Papadopoulos(7), Luis A. Díaz(7), Janine Ptak(7), Lisa Dobbyn(7), Natalie Silliman(7), Ian T. Jones(7), Suzanne Kosmider(6), Rachel Wong(6), Lara Lipton(6), Isaac Kinde(5), Ralph H. Hruban(5)