Piotr Zelazowski

Immunology and Microbiology · Life Sciences

19h-index1.3kcitations29works0.02yr avg
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Research Topics

T-cell and B-cell Immunology(7), Immune Response and Inflammation(6), Stress Responses and Cortisol(6), Immune Cell Function and Interaction(6), Immunotherapy and Immune Responses(5)

Publications29 total

Announcement / Erratum
Neuroendocrinology·2008Open Access
Distinct types of T-cell help for the induction of a humoral immune response to Streptococcus pneumoniae
Trends in Immunology·2001· 41 cited
NF-κB/p50 and NF-κB/c-Rel differentially regulate the activity of the 3′αE-hsl,2 enhancer in normal murine B cells in an activation-dependent manner
International Immunology·2000· 26 citedOpen Access
The p65 subunit of NF-kappa B is redundant with p50 during B cell proliferative responses, and is required for germline CH transcription and class switching to IgG3.
PubMed·1999· 56 cited
The p65 Subunit of NF-κB Is Redundant with p50 During B Cell Proliferative Responses, and Is Required for Germline CH Transcription and Class Switching to IgG3
The Journal of Immunology·1999· 44 citedOpen Access
B cells genetically deficient in the c-Rel transactivation domain have selective defects in germline CH transcription and Ig class switching
The Journal of Immunology·1997· 85 cited
Regulation of Ku expression in normal murine B cells by stimuli that promote switch recombination
The Journal of Immunology·1997· 28 cited
The Immunoglobulin Class Switch: Beyond “Accessibility”
Immunity·1997· 215 citedOpen Access
B cells lacking RelB are defective in proliferative responses, but undergo normal B cell maturation to Ig secretion and Ig class switching.
The Journal of Experimental Medicine·1996· 106 citedOpen Access
IL-10 selectively regulates murine Ig isotype switching
International Immunology·1996· 47 citedOpen Access
B cells from p50/NF-kappa B knockout mice have selective defects in proliferation, differentiation, germ-line CH transcription, and Ig class switching
The Journal of Immunology·1996· 299 citedOpen Access
Antigen receptor cross-linking differentially regulates germ-line CH ribonucleic acid expression in murine B cells.
The Journal of Immunology·1995· 27 cited
1,1′-Ethylidenebis[<i>L</i>-Tryptophan], a Contaminant Implicated in <i>L</i>-Try ptophan Eosinophilia Myalgia Syndrom, Suppresses mRNAE<i>x</i>pression of Hypothalamic Corticotropin-Refleasing Hormone in Lewis (LEW/N) Rat Brain
NeuroImmunoModulation·1994· 17 cited
Release of hypothalamic corticotropin-releasing hormone and arginine-vasopressin by interleukin 1β and αMSH: studies in rats with different susceptibility to inflammatory disease
Brain Research·1993· 44 cited
A decomposition product of a contaminant implicated in L-tryptophan eosinophilia myalgia syndrome affects spinal cord neuronal cell death and survival through stereospecific, maturation and partly interleukin-1-dependent mechanisms.
Journal of Pharmacology and Experimental Therapeutics·1993· 34 cited
Increased plasma concentrations, hypothalamic content, and in vitro release of arginine vasopressin in inflammatory disease-prone, hypothalamic corticotropin-releasing hormone-deficient Lewis rats.
Endocrinology·1992· 49 cited
In vitro Regulation of Pituitary ACTH Secretion in Inflammatory Disease Susceptible Lewis (LEW/N) and Inflammatory Disease Resistant Fischer (F344/N) Rats
Neuroendocrinology·1992· 32 cited
Announcement
Neuroendocrinology·1992Open Access
Melatonin‐Induced Suppression of Human Lymphocyte Natural Killer Activity in Vitro
Journal of Pineal Research·1989· 48 cited
Enhancement of human lymphocyte natural killer activity by somatostatin
Neuropeptides·1989· 17 cited
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Frequent Co-authors

Clifford M. Snapper(11), Marek Pawlikowski(7), H Stępień(7), Marilyn R. Kehry(4), Yi Shen(4), Philip W. Gold(4), Katarzyna Szewczyk(4), T Gościcka(4), F R Rosas(3), David Baltimore(3), George P. Chrousos(3), Klaus D. Döhler(3), Henryka Długońska(3), Mark A. Moorman(2), R Bravo(2), Bruce Horwitz(2), Karen Wolcott(2), Martin Scott(2), Vladimir K. Patchev(2), E Zelazowska(2)