Moon‐shong Tang

Biochemistry, Genetics and Molecular Biology · Life Sciences

30h-index5.0kcitations104works0.02yr avg
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Research Topics

DNA Repair Mechanisms(33), Bladder and Urothelial Cancer Treatments(29), Tissue Engineering and Regenerative Medicine(16), Carcinogens and Genotoxicity Assessment(15), Epigenetics and DNA Methylation(14)

Publications104 total

Supplementary Data from PKM2 Is Essential for Bladder Cancer Growth and Maintenance
2023Open Access
Supplementary Data from PKM2 Is Essential for Bladder Cancer Growth and Maintenance
2023Open Access
Supplementary Data from PKM2 Is Essential for Bladder Cancer Growth and Maintenance
2023Open Access
Data from PKM2 Is Essential for Bladder Cancer Growth and Maintenance
2023Open Access
Supplementary Figure from PKM2 Is Essential for Bladder Cancer Growth and Maintenance
2023Open Access
Data from PKM2 Is Essential for Bladder Cancer Growth and Maintenance
2023Open Access
Supplementary Data from PKM2 Is Essential for Bladder Cancer Growth and Maintenance
2023Open Access
Supplementary Figure from PKM2 Is Essential for Bladder Cancer Growth and Maintenance
2023Open Access
Supplemental Figure S3 from Oncogenic HRAS Activates Epithelial-to-Mesenchymal Transition and Confers Stemness to <i>p53</i>-Deficient Urothelial Cells to Drive Muscle Invasion of Basal Subtype Carcinomas
2023Open Access
Supplemental Figure S2 from Oncogenic HRAS Activates Epithelial-to-Mesenchymal Transition and Confers Stemness to <i>p53</i>-Deficient Urothelial Cells to Drive Muscle Invasion of Basal Subtype Carcinomas
2023Open Access
Data from Oncogenic HRAS Activates Epithelial-to-Mesenchymal Transition and Confers Stemness to <i>p53</i>-Deficient Urothelial Cells to Drive Muscle Invasion of Basal Subtype Carcinomas
2023Open Access
Supplemental Figure Legends from Oncogenic HRAS Activates Epithelial-to-Mesenchymal Transition and Confers Stemness to <i>p53</i>-Deficient Urothelial Cells to Drive Muscle Invasion of Basal Subtype Carcinomas
2023Open Access
Data from Oncogenic HRAS Activates Epithelial-to-Mesenchymal Transition and Confers Stemness to <i>p53</i>-Deficient Urothelial Cells to Drive Muscle Invasion of Basal Subtype Carcinomas
2023Open Access
Supplemental Figure S4 from Oncogenic HRAS Activates Epithelial-to-Mesenchymal Transition and Confers Stemness to <i>p53</i>-Deficient Urothelial Cells to Drive Muscle Invasion of Basal Subtype Carcinomas
2023Open Access
Supplemental Figure S2 from Oncogenic HRAS Activates Epithelial-to-Mesenchymal Transition and Confers Stemness to <i>p53</i>-Deficient Urothelial Cells to Drive Muscle Invasion of Basal Subtype Carcinomas
2023Open Access
Supplemental Figure S5 from Oncogenic HRAS Activates Epithelial-to-Mesenchymal Transition and Confers Stemness to <i>p53</i>-Deficient Urothelial Cells to Drive Muscle Invasion of Basal Subtype Carcinomas
2023Open Access
Supplemental Figure S3 from Oncogenic HRAS Activates Epithelial-to-Mesenchymal Transition and Confers Stemness to <i>p53</i>-Deficient Urothelial Cells to Drive Muscle Invasion of Basal Subtype Carcinomas
2023Open Access
Supplemental Table S1 from Oncogenic HRAS Activates Epithelial-to-Mesenchymal Transition and Confers Stemness to <i>p53</i>-Deficient Urothelial Cells to Drive Muscle Invasion of Basal Subtype Carcinomas
2023Open Access
Supplemental Figure S1 from Oncogenic HRAS Activates Epithelial-to-Mesenchymal Transition and Confers Stemness to <i>p53</i>-Deficient Urothelial Cells to Drive Muscle Invasion of Basal Subtype Carcinomas
2023Open Access
Supplemental Figure Legends from Oncogenic HRAS Activates Epithelial-to-Mesenchymal Transition and Confers Stemness to <i>p53</i>-Deficient Urothelial Cells to Drive Muscle Invasion of Basal Subtype Carcinomas
2023Open Access
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Frequent Co-authors

Zhaohui Feng(10), Wenwei Hu(9), Xue‐Ru Wu(7), Mikhail F. Denissenko(6), Gerd P. Pfeifer(6), Chuanshu Huang(6), William N. Rom(5), Annie Pao(4), Lung‐Chi Chen(4), Yi Zheng(4), Yu Hu(3), James X. Chen(3), He Wang(3), Max Costa(3), Feng He(3), Michael H. Patrick(3), Hyun‐Wook Lee(2), Yong Xia(2), Herbert Lepor(2), Frederick A. Beland(2)