Yaisa Andrews‐Zwilling

Annexon Biosciences (United States) — United States

Neuroscience · Life Sciences

13h-index2.3kcitations31works14.52yr avg
OpenAlex↗Google Scholar↗Lab page↗

Accepting Students?

No reports yet. Know if this professor is taking students?

Research Topics

Neuroinflammation and Neurodegeneration Mechanisms(10), Alzheimer's disease research and treatments(7), Neuroscience and Neuropharmacology Research(5), Systemic Lupus Erythematosus Research(3), Stress Responses and Cortisol(3)

Publications31 total

Targeting C1q prevents microglia-mediated synaptic removal in neuropathic pain
Nature Communications·2025· 16 citedOpen Access
Modified human mesenchymal stromal/stem cells restore cortical excitability after focal ischemic stroke in rats
Molecular Therapy·2024· 13 citedOpen Access
Classical Complement Activation in Lupus Nephritis Correlates with Disease Biomarkers: Results from Two Observational Studies
Journal of the American Society of Nephrology·2023
Results of Single-Arm, Phase 1b Study of Anti-C1q Treatment (ANX009) Show that the Classical Pathway Is a Key Driver of Complement Activation and Consumption in Patients with Active Lupus Nephritis
Journal of the American Society of Nephrology·2023
Pharmacokinetic and Target Engagement Measures of ANX007, an Anti-C1q Antibody Fragment, Following Intravitreal Administration in Nonhuman Primates
Investigative Ophthalmology & Visual Science·2023· 10 citedOpen Access
OP0232 HIGH PLASMA C4d/C4 IDENTIFIES LUPUS NEPHRITIS PATIENTS WITH DISEASE MEDIATED BY ACTIVATION OF THE CLASSICAL COMPLEMENT PATHWAY
Annals of the Rheumatic Diseases·2022Open Access
Inhibiting C1q Improves Compound Muscle Action Potential and Reduces Neuronal Damage in the SOD1G93A Mouse Model (P1-13.004)
Neurology·2022· 2 cited
Inhibition of C1q Reduces Nerve Damage as Measured by Neurofilament Light Chain in the HD R6/2 Mouse Model (S36.004)
Neurology·2022· 2 cited
Complement protein C1q is a therapeutic target for neuropathic pain
2021· 2 citedOpen Access
Complement factor C1q mediates sleep spindle loss and epileptic spikes after mild brain injury
Science·2021· 121 citedOpen Access
I11 Pharmacologic inhibition of the classical complement pathway enhances neuronal function and HD R6/2 mouse survival
2021· 1 cited
Pharmacokinetics and target engagement of intravitreal administration of ANX007, an anti-C1q antibody fragment, in nonhuman primates
Investigative Ophthalmology & Visual Science·2021· 3 citedOpen Access
Complement factor C1q mediates chronic neuron loss and inflammation post-brain injury
2020Open Access
Cell Therapy for Chronic Stroke
Stroke·2018· 102 citedOpen Access
Enhancing GABA Signaling during Middle Adulthood Prevents Age-Dependent GABAergic Interneuron Decline and Learning and Memory Deficits in ApoE4 Mice
Journal of Neuroscience·2016· 50 citedOpen Access
Inhibition of hilar GABAergic interneuron activity impaired spatial memory retrieval but not memory retention.
Figshare·2015Open Access
P2–037: Age‐dependent impairment of hilar GABAergic interneurons and cognitive function in Apolipoprotein E‐ε4 knock‐in mice is modified by sex
Alzheimer s & Dementia·2013
Apolipoprotein E4 Causes Age- and Sex-Dependent Impairments of Hilar GABAergic Interneurons and Learning and Memory Deficits in Mice
PLoS ONE·2012· 105 citedOpen Access
Hilar GABAergic Interneuron Activity Controls Spatial Learning and Memory Retrieval
PLoS ONE·2012· 100 citedOpen Access
P4‐253: Hilar GABAergic interneuron activity controls spatial learning and memory retrieval
Alzheimer s & Dementia·2012
page 1 of 2Next →

Frequent Co-authors

Yadong Huang(15), Ted Yednock(12), Vidhu Mathur(9), Daniel Zwilling(8), Nga Bien‐Ly(8), Seo Yeon Yoon(8), Karen Ring(7), Sethu Sankaranarayanan(7), Ellen Cahir-McFarland(6), Qin Xu(5), Aubrey Bernardo(5), Poojan Suri(5), Leslie M. Tong(4), Gang Li(4), Anatol C. Kreitzer(4), Noosha Yousefpour(4), David Walker(3), Jennifer K. Truong(3), Eric Hsieh(3), Jamie Louie(3)