Pranavi Koppula

The University of Texas MD Anderson Cancer Center — United States

Biochemistry, Genetics and Molecular Biology · Life Sciences

17h-index9.8kcitations31works100.02yr avg
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Research Topics

Cancer, Hypoxia, and Metabolism(15), Ferroptosis and cancer prognosis(12), RNA modifications and cancer(11), Epigenetics and DNA Methylation(10), Amino Acid Enzymes and Metabolism(7)

Publications31 total

Cell cycle arrest induces lipid droplet formation and confers ferroptosis resistance
Nature Communications·2024· 100 citedOpen Access
Supplementary Figure 1-8 from H2A Monoubiquitination Links Glucose Availability to Epigenetic Regulation of the Endoplasmic Reticulum Stress Response and Cancer Cell Death
2023Open Access
Supplementary Figure Legends from H2A Monoubiquitination Links Glucose Availability to Epigenetic Regulation of the Endoplasmic Reticulum Stress Response and Cancer Cell Death
2023Open Access
Table S1 from H2A Monoubiquitination Links Glucose Availability to Epigenetic Regulation of the Endoplasmic Reticulum Stress Response and Cancer Cell Death
2023Open Access
Data from H2A Monoubiquitination Links Glucose Availability to Epigenetic Regulation of the Endoplasmic Reticulum Stress Response and Cancer Cell Death
2023Open Access
Table S1 from H2A Monoubiquitination Links Glucose Availability to Epigenetic Regulation of the Endoplasmic Reticulum Stress Response and Cancer Cell Death
2023Open Access
Supplementary Figure 1-8 from H2A Monoubiquitination Links Glucose Availability to Epigenetic Regulation of the Endoplasmic Reticulum Stress Response and Cancer Cell Death
2023Open Access
Supplementary Figure Legends from H2A Monoubiquitination Links Glucose Availability to Epigenetic Regulation of the Endoplasmic Reticulum Stress Response and Cancer Cell Death
2023Open Access
Data from H2A Monoubiquitination Links Glucose Availability to Epigenetic Regulation of the Endoplasmic Reticulum Stress Response and Cancer Cell Death
2023Open Access
A targetable CoQ-FSP1 axis drives ferroptosis- and radiation-resistance in KEAP1 inactive lung cancers
Nature Communications·2022· 423 citedOpen Access
Thiol profiling in cancer cell lines by HPLC-mass spectrometry
STAR Protocols·2021· 5 citedOpen Access
Author Correction: DHODH-mediated ferroptosis defence is a targetable vulnerability in cancer
Nature·2021· 30 cited
KEAP1 deficiency drives glucose dependency and sensitizes lung cancer cells and tumors to GLUT inhibition
iScience·2021· 61 citedOpen Access
DHODH-mediated ferroptosis defence is a targetable vulnerability in cancer
Nature·2021· 1.7k citedOpen Access
Ferroptosis as a mechanism to mediate p53 function in tumor radiosensitivity
Oncogene·2021· 174 citedOpen Access
mTORC1 couples cyst(e)ine availability with GPX4 protein synthesis and ferroptosis regulation
Nature Communications·2021· 670 citedOpen Access
Cytochrome P450 reductase (POR) as a ferroptosis fuel
Protein & Cell·2021· 86 citedOpen Access
Keap1 Deficiency Drives Glucose Dependency and Sensitizes Lung Cancer Cells and Tumors to Glut Inhibition
SSRN Electronic Journal·2021· 1 citedOpen Access
Cystine transporter SLC7A11/xCT in cancer: ferroptosis, nutrient dependency, and cancer therapy
Protein & Cell·2020· 2.2k citedOpen Access
Abstract 2415: H2A monoubiquitination links glucose availability to epigenetic regulation of the endoplasmic reticulum stress induced cell death in cancer
Cancer Research·2020
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Frequent Co-authors

Boyi Gan(29), Yilei Zhang(25), Xiaoguang Liu(23), Hyemin Lee(16), Jiejun Shi(14), Wei Li(14), Guang Lei(12), Zhenna Xiao(11), Li Ma(10), Kellen Olszewski(8), Guang Lei(8), Weijie Cheng(8), Chao Mao(8), Chao Mao(7), Masha V. Poyurovsky(7), Li Zhuang(7), Bingliang Fang(6), Li Zhuang(4), Yuelong Yan(4), Li Zhuang(4)