Ian A. Blair

California University of Pennsylvania — United States

Biochemistry, Genetics and Molecular Biology · Life Sciences

80h-index28.6kcitations657works1.72yr avg
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Research Topics

Eicosanoids and Hypertension Pharmacology(67), Cancer, Lipids, and Metabolism(66), Metabolomics and Mass Spectrometry Studies(62), Cancer, Hypoxia, and Metabolism(58), Inflammatory mediators and NSAID effects(56)

Publications657 total

Author Correction: HMGB2-induced calreticulin translocation required for immunogenic cell death and ferroptosis of cancer cells are controlled by the nuclear exporter XPO1
Communications Biology·2025Open Access
Stable Isotope Labeling in Bacteria Enables Characterization and Quantification of Frataxin Protein in a Friedreich’s Ataxia Zebrafish Model
Analytical Chemistry·2025Open Access
Technical recommendations for analyzing oxylipins by liquid chromatography–mass spectrometry
Science Signaling·2025· 18 citedOpen Access
Individualized exercise and NAD+ precursor supplementation in Friedreich’s Ataxia: a randomized controlled trial
SSRN Electronic Journal·2025Open Access
Searching for enigmatic biomarkers to address a devastating degenerative disease
2025
HMGB2-induced calreticulin translocation required for immunogenic cell death and ferroptosis of cancer cells are controlled by the nuclear exporter XPO1
Communications Biology·2024· 13 citedOpen Access
Figure S4 from Bile Acid Metabolism Mediates Cholesterol Homeostasis and Promotes Tumorigenesis in Clear Cell Renal Cell Carcinoma
2024Open Access
Figure S7 from Bile Acid Metabolism Mediates Cholesterol Homeostasis and Promotes Tumorigenesis in Clear Cell Renal Cell Carcinoma
2024Open Access
Figure S6 from Bile Acid Metabolism Mediates Cholesterol Homeostasis and Promotes Tumorigenesis in Clear Cell Renal Cell Carcinoma
2024Open Access
Figure S3 from Bile Acid Metabolism Mediates Cholesterol Homeostasis and Promotes Tumorigenesis in Clear Cell Renal Cell Carcinoma
2024Open Access
Figure S2 from Bile Acid Metabolism Mediates Cholesterol Homeostasis and Promotes Tumorigenesis in Clear Cell Renal Cell Carcinoma
2024Open Access
Figure S2 from Bile Acid Metabolism Mediates Cholesterol Homeostasis and Promotes Tumorigenesis in Clear Cell Renal Cell Carcinoma
2024Open Access
Supplementary Data Text from Bile Acid Metabolism Mediates Cholesterol Homeostasis and Promotes Tumorigenesis in Clear Cell Renal Cell Carcinoma
2024Open Access
Figure S9 from Bile Acid Metabolism Mediates Cholesterol Homeostasis and Promotes Tumorigenesis in Clear Cell Renal Cell Carcinoma
2024Open Access
Figure S3 from Bile Acid Metabolism Mediates Cholesterol Homeostasis and Promotes Tumorigenesis in Clear Cell Renal Cell Carcinoma
2024Open Access
Figure S6 from Bile Acid Metabolism Mediates Cholesterol Homeostasis and Promotes Tumorigenesis in Clear Cell Renal Cell Carcinoma
2024Open Access
Supplementary Data Text from Bile Acid Metabolism Mediates Cholesterol Homeostasis and Promotes Tumorigenesis in Clear Cell Renal Cell Carcinoma
2024Open Access
Figure S8 from Bile Acid Metabolism Mediates Cholesterol Homeostasis and Promotes Tumorigenesis in Clear Cell Renal Cell Carcinoma
2024Open Access
Figure S4 from Bile Acid Metabolism Mediates Cholesterol Homeostasis and Promotes Tumorigenesis in Clear Cell Renal Cell Carcinoma
2024Open Access
Figure S1 from Bile Acid Metabolism Mediates Cholesterol Homeostasis and Promotes Tumorigenesis in Clear Cell Renal Cell Carcinoma
2024Open Access
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Frequent Co-authors

Nathaniel W. Snyder(11), Andrew J. Worth(7), Lili Guo(6), Clementina Mesaros(5), Seon Hwa Lee(5), Brian Keith(4), Kathryn E. Wellen(4), Michael C. Milone(3), Carl H. June(3), Joyce V. Lee(3), Alessandro Carrer(3), Sophie Trefely(3), Garret A. FitzGerald(3), T.M. Penning(3), Kavindra Nath(3), David Nelson(3), Dennis B. Leeper(3), Jerry D. Glickson(3), Dipti Mangal(2), Kenneth H. Yu(2)