Zoraida Diaz-Perez

University of Miami — United States

Medicine · Health Sciences

11h-index465citations18works3.72yr avg
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Research Topics

Autophagy in Disease and Therapy(6), Cardiomyopathy and Myosin Studies(5), Polyamine Metabolism and Applications(5), Cannabis and Cannabinoid Research(3), Sirtuins and Resveratrol in Medicine(3)

Publications18 total

Abstract LB060: NMNAT-mediated NAD+ metabolism promotes glioma growth by modulating p53 post-translational modifications and nuclear atypia
Cancer Research·2025
Nuclear NAD+ synthase nicotinamide mononucleotide adenylyltransferase 1 contributes to nuclear atypia and promotes glioma growth
Neuro-Oncology Advances·2025Open Access
Reduction of spermine synthase enhances autophagy to suppress Tau accumulation
Cell Death and Disease·2024· 11 citedOpen Access
Reduction of Spermine Synthase Suppresses Tau Accumulation Through Autophagy Modulation in Tauopathy
2023· 2 citedOpen Access
Phenylbutyrate modulates polyamine acetylase and ameliorates Snyder-Robinson syndrome in a Drosophila model and patient cells
JCI Insight·2022· 16 citedOpen Access
NMNAT promotes glioma growth through regulating post-translational modifications of P53 to inhibit apoptosis
eLife·2021· 24 citedOpen Access
Author response: NMNAT promotes glioma growth through regulating post-translational modifications of P53 to inhibit apoptosis
2021Open Access
NMNAT promotes glioma growth through regulating post-translational modifications of p53 to inhibit apoptosis
2021· 2 citedOpen Access
Exposure to Aerosolized Algal Toxins in South Florida Increases Short- and Long-Term Health Risk in Drosophila Model of Aging
Toxins·2020· 25 citedOpen Access
Nmnat restores neuronal integrity by neutralizing mutant Huntingtin aggregate-induced progressive toxicity
Proceedings of the National Academy of Sciences·2019· 39 citedOpen Access
Publisher Correction: Spermine synthase deficiency causes lysosomal dysfunction and oxidative stress in models of Snyder-Robinson syndrome
Nature Communications·2018Open Access
Spermine synthase deficiency causes lysosomal dysfunction and oxidative stress in models of Snyder-Robinson syndrome
Nature Communications·2017· 99 citedOpen Access
Defining Disease, Diagnosis, and Translational Medicine within a Homeostatic Perturbation Paradigm: The National Institutes of Health Undiagnosed Diseases Program Experience
Frontiers in Medicine·2017· 28 citedOpen Access
Generation and Functional Characterization of Knock-in Mice Harboring the Cardiac Troponin I-R21C Mutation Associated with Hypertrophic Cardiomyopathy
Journal of Biological Chemistry·2011· 40 citedOpen Access
Protein Kinase a Phosphorylation of Cardiac Troponin I Prevents Cardiac Hypertrophy in Mice
Biophysical Journal·2010Open Access
Fast skeletal muscle regulatory light chain is required for fast and slow skeletal muscle development
The FASEB Journal·2007· 49 cited
Prolonged Ca2+ and Force Transients in Myosin RLC Transgenic Mouse Fibers Expressing Malignant and Benign FHC Mutations
Journal of Molecular Biology·2006· 63 cited
The E22K mutation of myosin RLC that causes familial hypertrophic cardiomyopathy increases calcium sensitivity of force and ATPase in transgenic mice
Journal of Cell Science·2005· 67 cited

Frequent Co-authors

R. Grace Zhai(13), Yi Zhu(11), Xianzun Tao(8), Chong Li(7), Jiaqi Liu(7), Hongbo Wang(5), Jennifer M. Brazill(4), Yingcai Wang(4), Kai Ruan(4), Christofer Bello(3), May Christine V. Malicdan(3), William A. Gahl(3), Cornelius F. Boerkoel(3), Danuta Szczesna‐Cordary(3), Georgianna Guzman(3), James D. Potter(3), Priyamvada Rai(3), Jackson R. Foley(3), Tracy Murray Stewart(3), Robert A. Casero(3)