Jane Fridlyand

Systems Analytics (United States) — United States

Biochemistry, Genetics and Molecular Biology · Life Sciences

46h-index20.1kcitations202works1.62yr avg
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Research Topics

Cancer Genomics and Diagnostics(64), Cancer Cells and Metastasis(59), Genomic variations and chromosomal abnormalities(31), Gene expression and cancer classification(21), Breast Cancer Treatment Studies(19)

Publications202 total

Evaluations of Backfill Strategies in Dose Optimization through Simulation Studies for Phase I Trials in Oncology
Statistics in Biopharmaceutical Research·2025· 2 cited
Abstract 2514: Refining oncology dose selection: A framework for tailored dose optimization in the era of targeted and immunotherapies
Cancer Research·2025
Oncology Dose Optimization: Tailored Approaches to Different Molecular Classes
Clinical Pharmacology & Therapeutics·2025· 3 citedOpen Access
Model Generalizability Considerations in Development and Evaluation of SaMD
2024Open Access
Dose Optimization for Novel Oncology Agents: Design Options and Strategies
Statistics in Biopharmaceutical Research·2024· 3 cited
Abstract 5705: Digital pathology based prognostic & predictive biomarkers in metastatic non-small cell lung cancer
Cancer Research·2023· 1 cited
Supplementary Figures S1-S2 from Amplification of <i>PVT1</i> Contributes to the Pathophysiology of Ovarian and Breast Cancer
2023Open Access
Primary aCGH data and supplementary tables from Chromosomal Instability in Microsatellite-Unstable and Stable Colon Cancer
2023Open Access
Primary aCGH data and supplementary tables from Chromosomal Instability in Microsatellite-Unstable and Stable Colon Cancer
2023Open Access
Data from Amplification of <i>PVT1</i> Contributes to the Pathophysiology of Ovarian and Breast Cancer
2023Open Access
Supplementary Tables S1-S4 from Amplification of <i>PVT1</i> Contributes to the Pathophysiology of Ovarian and Breast Cancer
2023Open Access
Supplementary Methods from Phosphoinositide 3-Kinase (PI3K) Pathway Alterations Are Associated with Histologic Subtypes and Are Predictive of Sensitivity to PI3K Inhibitors in Lung Cancer Preclinical Models
2023Open Access
Supplementary Table 2 from Phosphoinositide 3-Kinase (PI3K) Pathway Alterations Are Associated with Histologic Subtypes and Are Predictive of Sensitivity to PI3K Inhibitors in Lung Cancer Preclinical Models
2023Open Access
Supplementary Tables S1-S4 from Amplification of <i>PVT1</i> Contributes to the Pathophysiology of Ovarian and Breast Cancer
2023Open Access
Supplementary Table 1 from Phosphoinositide 3-Kinase (PI3K) Pathway Alterations Are Associated with Histologic Subtypes and Are Predictive of Sensitivity to PI3K Inhibitors in Lung Cancer Preclinical Models
2023Open Access
Data from Phosphoinositide 3-Kinase (PI3K) Pathway Alterations Are Associated with Histologic Subtypes and Are Predictive of Sensitivity to PI3K Inhibitors in Lung Cancer Preclinical Models
2023Open Access
Data from Chromosomal Instability in Microsatellite-Unstable and Stable Colon Cancer
2023Open Access
Supplementary Methods from Phosphoinositide 3-Kinase (PI3K) Pathway Alterations Are Associated with Histologic Subtypes and Are Predictive of Sensitivity to PI3K Inhibitors in Lung Cancer Preclinical Models
2023Open Access
Supplementary Table 1 from Phosphoinositide 3-Kinase (PI3K) Pathway Alterations Are Associated with Histologic Subtypes and Are Predictive of Sensitivity to PI3K Inhibitors in Lung Cancer Preclinical Models
2023Open Access
Supplementary Figures S1-S2 from Amplification of <i>PVT1</i> Contributes to the Pathophysiology of Ovarian and Breast Cancer
2023Open Access
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Frequent Co-authors

Donna G. Albertson(21), Daniel Pinkel(17), Ajay N. Jain(12), Joe W. Gray(10), Antoine M. Snijders(8), Frederic M. Waldman(7), Sandy DeVries(6), Ritu Roydasgupta(6), Sandrine Dudoit(5), Boris C. Bastian(5), Koei Chin(4), Taku A. Tokuyasu(4), Shanaz H. Dairkee(4), Peter R. Carroll(4), Richard M. Neve(3), Terence P. Speed(3), Anna Lapuk(3), Wen-Lin Kuo(3), Karen Chew(3), Jian‐Hua Mao(3)