Timothy A. Chan

Cleveland Clinic — United States

Medicine · Health Sciences

131h-index110.2kcitations1.0kworks3.62yr avg
OpenAlex↗ORCID↗Google Scholar↗Lab page↗

Accepting Students?

No reports yet. Know if this professor is taking students?

Research Topics

Cancer Immunotherapy and Biomarkers(246), Cancer Genomics and Diagnostics(173), Renal cell carcinoma treatment(116), Glioma Diagnosis and Treatment(108), Immunotherapy and Immune Responses(108)

Publications1.0k total

Supplementary Table S1 from KDM6A Loss Triggers an Epigenetic Switch That Disrupts Urothelial Differentiation and Drives Cell Proliferation in Bladder Cancer
2025Open Access
Supplementary Figure Legends from KDM6A Loss Triggers an Epigenetic Switch That Disrupts Urothelial Differentiation and Drives Cell Proliferation in Bladder Cancer
2025Open Access
Supplementary Data 1 from KDM6A Loss Triggers an Epigenetic Switch That Disrupts Urothelial Differentiation and Drives Cell Proliferation in Bladder Cancer
2025Open Access
Supplementary Figure 7 from KDM6A Loss Triggers an Epigenetic Switch That Disrupts Urothelial Differentiation and Drives Cell Proliferation in Bladder Cancer
2025Open Access
Supplementary Figure 1 from KDM6A Loss Triggers an Epigenetic Switch That Disrupts Urothelial Differentiation and Drives Cell Proliferation in Bladder Cancer
2025Open Access
Supplementary Figure 8 from KDM6A Loss Triggers an Epigenetic Switch That Disrupts Urothelial Differentiation and Drives Cell Proliferation in Bladder Cancer
2025Open Access
Supplementary Figure 5 from KDM6A Loss Triggers an Epigenetic Switch That Disrupts Urothelial Differentiation and Drives Cell Proliferation in Bladder Cancer
2025Open Access
Supplementary Figure 6 from KDM6A Loss Triggers an Epigenetic Switch That Disrupts Urothelial Differentiation and Drives Cell Proliferation in Bladder Cancer
2025Open Access
Supplementary Figure 4 from KDM6A Loss Triggers an Epigenetic Switch That Disrupts Urothelial Differentiation and Drives Cell Proliferation in Bladder Cancer
2025Open Access
Supplementary Figure 3 from KDM6A Loss Triggers an Epigenetic Switch That Disrupts Urothelial Differentiation and Drives Cell Proliferation in Bladder Cancer
2025Open Access
Supplementary Figure 2 from KDM6A Loss Triggers an Epigenetic Switch That Disrupts Urothelial Differentiation and Drives Cell Proliferation in Bladder Cancer
2025Open Access
Supplementary Data 2 from KDM6A Loss Triggers an Epigenetic Switch That Disrupts Urothelial Differentiation and Drives Cell Proliferation in Bladder Cancer
2025Open Access
The SLC1A1/EAAT3 dicarboxylic amino acid transporter is an epigenetically dysregulated nutrient carrier that sustains oncogenic metabolic programs
Nature Communications·2025· 1 citedOpen Access
EWSR1 fusion neoantigen vaccine for the treatment of recurrent Ewing sarcoma 9402
The Journal of Immunology·2025Open Access
1144 Total intratumoral bacteria abundance drives immunosuppression and immunotherapy resistance
Regular and Young Investigator Award Abstracts·2025Open Access
RADT-15. CLINICAL OUTCOMES OF PATIENTS WITH BRAIN METASTASES FROM SMALL CELL LUNG CANCER TREATED WITH STEREOTACTIC RADIOSURGERY
Neuro-Oncology·2025Open Access
Hypoxia and tumor associated macrophages shape response to immune and angiogenesis-targeted therapies in metastatic clear cell renal cell carcinoma 3738
The Journal of Immunology·2025Open Access
S4832 Parastomal Varices: When Bleeding Demands a Shunt
The American Journal of Gastroenterology·2025
S3635 Acute Pancreatitis Following Transjugular Liver Biopsy: A Rare Complication
The American Journal of Gastroenterology·2025
34Hypoxia and immune suppression shapes therapeutic outcomes and metastases in clear cell renal cell carcinoma
The Oncologist·2025Open Access
page 1 of 51Next →

Frequent Co-authors

Nadeem Riaz(14), Vladimir Makarov(13), Luc G.T. Morris(12), Jedd D. Wolchok(10), Ian Ganly(8), Fengshen Kuo(8), Taha Merghoub(7), A. Ari Hakimi(7), Naiyer A. Rizvi(6), Jonathan J. Havel(6), Alexis Desrichard(6), David B. Solit(6), Diego Chowell(6), Alexandra Snyder(5), Chris Sander(5), Matthew D. Hellmann(4), Robert Samstein(4), Ahmet Zehir(4), Martin H. Voss(4), Ingo K. Mellinghoff(4)