R. Oliver Goral

National Institutes of Health — United States

Neuroscience · Life Sciences

7h-index664citations20works1.32yr avg
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Research Topics

Neuroscience and Neuropharmacology Research(8), Ion channel regulation and function(8), Nicotinic Acetylcholine Receptors Study(6), Cellular transport and secretion(4), Pain Mechanisms and Treatments(3)

Publications20 total

Mesoscopic analysis of GABAergic marker expression in acetylcholine neurons in the whole mouse brain
2025Open Access
Erythematous-purpuric lesions in a one-year-old child revealing a case of acute hemorrhagic edema of childhood.
Dermatology Online Journal·2025Open Access
Acetylcholine Neurons Become Cholinergic during Three Time Windows in the Developing Mouse Brain
eNeuro·2024· 4 citedOpen Access
Loss of GABA co-transmission from cholinergic neurons impairs behaviors related to hippocampal, striatal, and medial prefrontal cortex functions
Frontiers in Behavioral Neuroscience·2022· 6 citedOpen Access
Loss of GABA co-transmission from cholinergic neurons impairs behaviors related to hippocampal, striatal, and medial prefrontal cortex functions
2022Open Access
Presynaptic development is controlled by the core active zone proteins CAST/ELKS
The Journal of Physiology·2020· 28 citedOpen Access
Presynaptic development is controlled by the core active zone proteins CAST/ELKS
2019· 5 citedOpen Access
CaV2.1 α1 Subunit Expression Regulates Presynaptic CaV2.1 Abundance and Synaptic Strength at a Central Synapse
Neuron·2018· 59 citedOpen Access
CAST/ELKS Proteins Control Voltage-Gated Ca2+ Channel Density and Synaptic Release Probability at a Mammalian Central Synapse
Cell Reports·2018· 85 citedOpen Access
A novel region in the CaV2.1 α1 subunit C-terminus regulates fast synaptic vesicle fusion and vesicle docking at the mammalian presynaptic active zone
eLife·2017· 52 citedOpen Access
Author response: A novel region in the CaV2.1 α1 subunit C-terminus regulates fast synaptic vesicle fusion and vesicle docking at the mammalian presynaptic active zone
2017Open Access
Cold-aggravated pain in humans caused by a hyperactive NaV1.9 channel mutant
Nature Communications·2015· 87 citedOpen Access
Heterologous expression of NaV1.9 chimeras in various cell systems
Pflügers Archiv - European Journal of Physiology·2015· 35 cited
Increased Activity of Nav1.9 Sodium Channels Causes Loss of Pain Perception
Biophysical Journal·2014Open Access
A de novo gain-of-function mutation in SCN11A causes loss of pain perception
Nature Genetics·2013· 302 cited
[Morphology and physiology of the autonomic nervous system of the liver].
PubMed·1975
Effect of ethacrynic acid on water transfer and sodium transfer across human peritoneal membrane.
PubMed·1975· 1 cited
[Torsion of the cecum].
PubMed·1968
[Accessory vascularization of the liver].
PubMed·1965
[Gastric polypi (the problem of malignant degeneration)].
PubMed·1961

Frequent Co-authors

Naomi Kamasawa(6), Samuel Young(6), Matthias Lübbert(5), Enrico Leipold(4), Stefan H. Heinemann(4), Connon I. Thomas(4), Debbie Guerrero‐Given(4), Travis Putzke(4), Patricia W. Lamb(4), Jerrel L. Yakel(4), Christian A. Hübner(3), Ingo Kurth(3), Wei Dong(3), Tamara Radulović(3), Mónica S. Montesinos(3), Toshihisa Ohtsuka(3), Rachel Satterfield(3), Janine Altmüller(2), Peter Nürnberg(2), Joachim Weis(2)