F Schmid

Landeskrankenhaus Feldkirch — Austria

Biochemistry, Genetics and Molecular Biology · Life Sciences

18h-index944citations58works0.02yr avg
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Research Topics

Genetic and Kidney Cyst Diseases(8), Cardiovascular Disease and Adiposity(7), Lipoproteins and Cardiovascular Health(5), Renal and related cancers(4), Diabetes, Cardiovascular Risks, and Lipoproteins(3)

Publications58 total

Conservative Treatment of Spontaneous Neonatal Aortic Thrombosis: A Case Study Expanding the Evidence Base
Klinische Pädiatrie·2025
Cilia and Flagella
Elsevier eBooks·2022· 2 cited
275 TRANSURETHRAL INJECTION OF AUTOLOGOUS MUSCLE PRECURSOR CELLS FOR THE TREATMENT OF FEMALE STRESS URINARY INCONTINENCE – A PROSPECTIVE AND RANDOMIZED PHASE I CLINICAL TRIAL
Continence·2022Open Access
Interferon-stimulated gene 15 accelerates replication fork progression inducing chromosomal breakage
The Journal of Cell Biology·2020· 73 citedOpen Access
Floating kidneys associated with Eisenmenger syndrome
Kidney International·2018
Histone Ubiquitination by the DNA Damage Response Is Required for Efficient DNA Replication in Unperturbed S Phase
Molecular Cell·2018· 102 citedOpen Access
IFT20 modulates ciliary PDGFRα signaling by regulating the stability of Cbl E3 ubiquitin ligases
The Journal of Cell Biology·2017· 63 citedOpen Access
Cilia and Flagella
Elsevier eBooks·2015· 1 cited
Coordination of TGFβ/BMP signaling is associated with the primary cilium
Cilia·2015· 9 citedOpen Access
Expression of the oncogenic, constitutive active Platelet-Derived Growth Factor Receptor alpha D842V mutant induces loss of primary cilia
Cilia·2015Open Access
Analgesia via blockade of NGF/TrkA signaling does not influence fracture healing in mice
Journal of Orthopaedic Research®·2015· 42 citedOpen Access
PDGFRβ and oncogenic, mutant PDGFRα D842V promote disassembly of primary cilia by a PLCγ and AURKA dependent mechanism
Journal of Cell Science·2015· 35 cited
Metabolisches Syndrom und antihypertensive Therapie
Journal für Kardiologie (Krause & Pachernegg GmbH)·2013Open Access
A Gene Therapeutic Approach to Correct Splice Defects with Modified U1 and U6 snRNPs
Human Gene Therapy·2012· 31 citedOpen Access
What range of pace-sense-compensation should be provided in biventricular pacing systems for heart failure?
Biomedizinische Technik/Biomedical Engineering·2012
Gene Therapy To Rescue Splice Defects In Eye Diseases Using U1 snRNA
Investigative Ophthalmology & Visual Science·2011
U1 snRNA-mediated gene therapeutic correction of splice defects caused by an exceptionally mild BBS mutation
Human Mutation·2011· 61 cited
Gene Therapeutic Approach Using Mutation-adapted U1 snRNA to Correct a RPGR Splice Defect in Patient-derived Cells
Molecular Therapy·2011· 66 citedOpen Access
Identification and treatment of splice defects in ciliary genes RPGR and BBS1 causing Retinitis pigmentosa
Zurich Open Repository and Archive (University of Zurich)·2011Open Access
MS228 TYPE 2 DIABETES AND THE CORONARY ANGIOGRAPHIC STATE ARE MUTUALLY INDEPENDENT PREDICTORS OF FUTURE VASCULAR EVENTS AMONG ANGIOGRAPHIED CORONARY PATIENTS
Atherosclerosis Supplements·2010
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Frequent Co-authors

Thomas Marte(19), Heinz Drexel(18), Stefan Aczél(17), Philipp Rein(13), Christoph H. Saely(12), Lorena Koch(11), P. Langer(11), Guenter Hoefle(10), C.H. Saely(7), Esther Glaus(6), Wolfgang Berger(6), John Neidhardt(6), Lorenz Risch(6), Lotte B. Pedersen(5), Søren T. Christensen(5), Axel Muendlein(4), Linda Koch(4), Franziska Walser(2), Maria Chiara Raso(2), Lorenza Penengo(2)