María J. Andrés‐Manzano

MACOM (United States) — United States

Biochemistry, Genetics and Molecular Biology · Life Sciences

15h-index886citations25works8.52yr avg
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Research Topics

Nuclear Structure and Function(16), RNA Research and Splicing(11), PARP inhibition in cancer therapy(5), Phagocytosis and Immune Regulation(3), Atherosclerosis and Cardiovascular Diseases(3)

Publications25 total

Lamin A/C Expression in Hematopoietic Cells Declines During Human Aging and Constrains Atherosclerosis in Mice
Arteriosclerosis Thrombosis and Vascular Biology·2025· 2 citedOpen Access
Endothelial cell‐specific progerin expression does not cause cardiovascular alterations and premature death
Aging Cell·2024· 5 citedOpen Access
Endothelial YAP/TAZ activation promotes atherosclerosis in a mouse model of Hutchinson-Gilford progeria syndrome
Journal of Clinical Investigation·2024· 14 citedOpen Access
Endothelial-to-Mesenchymal Transition Contributes to Accelerated Atherosclerosis in Hutchinson-Gilford Progeria Syndrome
Circulation·2024· 19 citedOpen Access
Endothelial-to-mesenchymal transition contributes to accelerated atherosclerosis during premature aging
Atherosclerosis·2024Open Access
Exacerbated atherosclerosis in progeria is prevented by progerin elimination in vascular smooth muscle cells but not endothelial cells
Proceedings of the National Academy of Sciences·2024· 11 citedOpen Access
Atherosclerotic plaque development in mice is enhanced by myeloid ZEB1 downregulation
Nature Communications·2023· 10 citedOpen Access
Lamin A/C Ablation Restricted to Vascular Smooth Muscle Cells, Cardiomyocytes, and Cardiac Fibroblasts Causes Cardiac and Vascular Dysfunction
International Journal of Molecular Sciences·2023· 1 citedOpen Access
Lamin A/C Ablation in Vascular Smooth Muscle Cells, Cardiomyocytes, and Cardiac Fibroblasts Causes Cardiac and Vascular Dysfunction: Role in the Pathophysiology of LMNA-Related Dilated Cardiomyopathies
Preprints.org·2023Open Access
Coronary and carotid artery dysfunction and KV7 overexpression in a mouse model of Hutchinson-Gilford progeria syndrome
GeroScience·2023· 1 citedOpen Access
Lamin A/C expression in hematopoietic cells regulates mouse atherosclerosis: A potential mechanism contributing to age-dependent atherosclerosis
Atherosclerosis·2022Open Access
Cardiovascular Progerin Suppression and Lamin A Restoration Rescue Hutchinson-Gilford Progeria Syndrome
Circulation·2021· 42 citedOpen Access
Paclitaxel mitigates structural alterations and cardiac conduction system defects in a mouse model of Hutchinson–Gilford progeria syndrome
Cardiovascular Research·2021· 22 citedOpen Access
Premature Vascular Aging with Features of Plaque Vulnerability in an Atheroprone Mouse Model of Hutchinson–Gilford Progeria Syndrome with Ldlr Deficiency
Cells·2020· 23 citedOpen Access
Identification of common cardiometabolic alterations and deregulated pathways in mouse and pig models of aging
Aging Cell·2020· 18 citedOpen Access
Vascular Smooth Muscle Cell-Specific Progerin Expression Provokes Contractile Impairment in a Mouse Model of Hutchinson-Gilford Progeria Syndrome that Is Ameliorated by Nitrite Treatment
Cells·2020· 32 citedOpen Access
Disruption of the CCL1-CCR8 axis inhibits vascular Treg recruitment and function and promotes atherosclerosis in mice
Journal of Molecular and Cellular Cardiology·2019· 46 cited
Generation and characterization of a novel knockin minipig model of Hutchinson-Gilford progeria syndrome
Cell Discovery·2019· 62 citedOpen Access
Progerin accelerates atherosclerosis by inducing endoplasmic reticulum stress in vascular smooth muscle cells
EMBO Molecular Medicine·2019· 117 citedOpen Access
miR-146a deficiency in hematopoietic cells is not involved in the development of atherosclerosis
PLoS ONE·2018· 19 citedOpen Access
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Frequent Co-authors

Vicente Andrés(24), Pilar Gonzalo(15), Beatriz Dorado(11), Cristina González‐Gómez(10), Magda R. Hamczyk(8), Rosa M. Nevado(7), Víctor Fanjul(6), Carlos López-Otı́n(5), Ana Barettino(5), Álvaro Macías(5), Alberto Del Monte-Monge(5), Ignacio Benedicto(5), Carla Espinós‐Estévez(4), Paula Nogales(3), Carlos Torroja(3), Fátima Sánchez‐Cabo(3), Ana Dopazo(3), Rosa María Carmona(3), Miguel de la Fuente‐Pérez(3), Ricardo Villa‐Bellosta(2)