Noriaki Tanaka

The University of Texas MD Anderson Cancer Center — United States

Biochemistry, Genetics and Molecular Biology · Life Sciences

64h-index15.4kcitations682works1.02yr avg
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Research Topics

Virus-based gene therapy research(92), Cancer Research and Treatments(71), Cancer-related Molecular Pathways(52), Liver physiology and pathology(49), Organ Transplantation Techniques and Outcomes(49)

Publications681 total

NOTCH1 Acts as a Tumor Suppressor That Induces Early Differentiation in Head and Neck Cancer
2025· 1 citedOpen Access
Supplementary Figure S6 & Supplementary S6 Legend from Combined Inhibition of Rad51 and Wee1 Enhances Cell Killing in HNSCC Through Induction of Apoptosis Associated With Excessive DNA Damage and Replication Stress
2023Open Access
Supplementary Figure S4 & Supplementary S4 Legend from Combined Inhibition of Rad51 and Wee1 Enhances Cell Killing in HNSCC Through Induction of Apoptosis Associated With Excessive DNA Damage and Replication Stress
2023Open Access
Supplementary Figure S6 & Supplementary S6 Legend from Combined Inhibition of Rad51 and Wee1 Enhances Cell Killing in HNSCC Through Induction of Apoptosis Associated With Excessive DNA Damage and Replication Stress
2023Open Access
Supplementary Figure S7 & Supplementary S7 Legend from Combined Inhibition of Rad51 and Wee1 Enhances Cell Killing in HNSCC Through Induction of Apoptosis Associated With Excessive DNA Damage and Replication Stress
2023Open Access
Supplementary Figure S7 & Supplementary S7 Legend from Combined Inhibition of Rad51 and Wee1 Enhances Cell Killing in HNSCC Through Induction of Apoptosis Associated With Excessive DNA Damage and Replication Stress
2023Open Access
Supplementary Figures S1-S3 & Supplementary S1-S3 Legends from Combined Inhibition of Rad51 and Wee1 Enhances Cell Killing in HNSCC Through Induction of Apoptosis Associated With Excessive DNA Damage and Replication Stress
2023Open Access
Supplementary Figure S5 & Supplementary S5 Legend from Combined Inhibition of Rad51 and Wee1 Enhances Cell Killing in HNSCC Through Induction of Apoptosis Associated With Excessive DNA Damage and Replication Stress
2023Open Access
Supplementary Table S1 from Combined Inhibition of Rad51 and Wee1 Enhances Cell Killing in HNSCC Through Induction of Apoptosis Associated With Excessive DNA Damage and Replication Stress
2023Open Access
Data from Combined Inhibition of Rad51 and Wee1 Enhances Cell Killing in HNSCC Through Induction of Apoptosis Associated With Excessive DNA Damage and Replication Stress
2023Open Access
Supplementary Figure S4 & Supplementary S4 Legend from Combined Inhibition of Rad51 and Wee1 Enhances Cell Killing in HNSCC Through Induction of Apoptosis Associated With Excessive DNA Damage and Replication Stress
2023Open Access
Supplementary Figures S1-S3 & Supplementary S1-S3 Legends from Combined Inhibition of Rad51 and Wee1 Enhances Cell Killing in HNSCC Through Induction of Apoptosis Associated With Excessive DNA Damage and Replication Stress
2023Open Access
Supplementary Materials and Methods from Combined Inhibition of Rad51 and Wee1 Enhances Cell Killing in HNSCC Through Induction of Apoptosis Associated With Excessive DNA Damage and Replication Stress
2023Open Access
Supplementary Table S1 from Combined Inhibition of Rad51 and Wee1 Enhances Cell Killing in HNSCC Through Induction of Apoptosis Associated With Excessive DNA Damage and Replication Stress
2023Open Access
Supplementary Materials and Methods from Combined Inhibition of Rad51 and Wee1 Enhances Cell Killing in HNSCC Through Induction of Apoptosis Associated With Excessive DNA Damage and Replication Stress
2023Open Access
Data from Combined Inhibition of Rad51 and Wee1 Enhances Cell Killing in HNSCC Through Induction of Apoptosis Associated With Excessive DNA Damage and Replication Stress
2023Open Access
Supplementary Figure S5 & Supplementary S5 Legend from Combined Inhibition of Rad51 and Wee1 Enhances Cell Killing in HNSCC Through Induction of Apoptosis Associated With Excessive DNA Damage and Replication Stress
2023Open Access
Supplementary Figure 2 from Preclinical Evaluation of Differentially Targeting Dual Virotherapy for Human Solid Cancer
2023Open Access
Supplementary Figure S8 from Wee-1 Kinase Inhibition Sensitizes High-Risk HPV<sup>+</sup> HNSCC to Apoptosis Accompanied by Downregulation of MCl-1 and XIAP Antiapoptotic Proteins
2023Open Access
Supplementary Figure Legends from Wee-1 Kinase Inhibition Sensitizes High-Risk HPV<sup>+</sup> HNSCC to Apoptosis Accompanied by Downregulation of MCl-1 and XIAP Antiapoptotic Proteins
2023Open Access
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Frequent Co-authors

Toshiyoshi Fujiwara(14), Naoya Kobayashi(10), Yoshio Naomoto(8), Shunsuke Kagawa(7), Hirofumi Noguchi(6), Nagahide Matsubara(6), Teru Okitsu(5), Futoshi Uno(5), Yasuo Urata(5), Karen A. Westerman(4), Masakiyo Sakaguchi(4), Philippe Leboulch(4), Satoru Kyo(4), Takeshi Nagasaka(4), Hiromi Sasamoto(4), Jorge David Rivas‐Carrillo(4), Nalu Navarro–Álvarez(4), Munenori Takaoka(4), Minoru Haisa(4), Takuya Fukazawa(4)