Edwin J. Weeber

University of South Florida — United States

Neuroscience · Life Sciences

61h-index12.0kcitations148works4.52yr avg
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Research Topics

Neuroscience and Neuropharmacology Research(33), Genetic Syndromes and Imprinting(32), Genetics and Neurodevelopmental Disorders(25), Neurogenesis and neuroplasticity mechanisms(24), Epigenetics and DNA Methylation(23)

Publications148 total

A preclinical pig model of Angelman syndrome mirrors the early developmental trajectory of the human condition
Proceedings of the National Academy of Sciences·2025· 3 citedOpen Access
DISC1 and reelin interact to alter cognition, inhibition, and neurogenesis in a novel mouse model of schizophrenia
Frontiers in Cellular Neuroscience·2024· 6 citedOpen Access
DISC1 and Reelin interact to alter cognition, inhibition, and neurogenesis in a novel mouse model of schizophrenia
Harvard Dataverse·2023Open Access
Central repeat fragment of reelin leads to active reelin intracellular signaling and rescues cognitive deficits in a mouse model of reelin deficiency
Cellular Signalling·2023· 5 cited
A novel Reelin construct, R36, recovered behavioural deficits in the heterozygous reeler mouse
European Journal of Neuroscience·2023· 5 cited
An ASO therapy for Angelman syndrome that targets an evolutionarily conserved region at the start of the <i>UBE3A-AS</i> transcript
Science Translational Medicine·2023· 81 cited
Author response for "A novel Reelin construct, R36, recovered behavioral deficits in the heterozygous reeler mouse"
2023
Reelin and APP Cooperatively Modulate Dendritic Spine Formation <i>In Vitro</i> and <i>In Vivo</i>
Experimental Neurobiology·2023· 8 citedOpen Access
Correction to: Slingshot-Cofilin activation mediates mitochondrial and synaptic dysfunction via A β ligation to β1-integrin conformers
Cell Death and Differentiation·2022Open Access
Reelin central fragment supplementation improves cognitive deficits in a mouse model of Fragile X Syndrome
Experimental Neurology·2022· 5 cited
Recovery of Angelman syndrome rat deficits with UBE3A protein supplementation
Molecular and Cellular Neuroscience·2022· 6 cited
Accumulation of C-terminal cleaved tau is distinctly associated with cognitive deficits, synaptic plasticity impairment, and neurodegeneration in aged mice
GeroScience·2021· 12 citedOpen Access
Early Developmental EEG and Seizure Phenotypes in a Full Gene Deletion of Ubiquitin Protein Ligase E3A Rat Model of Angelman Syndrome
eNeuro·2021· 29 citedOpen Access
Identification of <scp>UBE3A</scp> Protein in <scp>CSF</scp> and Extracellular Space of the Hippocampus Suggest a Potential Novel Function in Synaptic Plasticity
Autism Research·2021· 12 cited
Quantitative EEG Analysis in Angelman Syndrome: Candidate Method for Assessing Therapeutics
Clinical EEG and Neuroscience·2020· 26 cited
Two forms of CX3CL1 display differential activity and rescue cognitive deficits in CX3CL1 knockout mice
Journal of Neuroinflammation·2020· 61 citedOpen Access
Translational outcomes in a full gene deletion of ubiquitin protein ligase E3A rat model of Angelman syndrome
Translational Psychiatry·2020· 61 citedOpen Access
Generation of a Novel Rat Model of Angelman Syndrome with a Complete <i>Ube3a</i> Gene Deletion
Autism Research·2020· 33 citedOpen Access
Evaluation of the safety and tolerability of a nutritional Formulation in patients with ANgelman Syndrome (FANS): study protocol for a randomized controlled trial
Trials·2020· 12 citedOpen Access
Evaluation of the Safety and Tolerability of a Nutritional Formulation in Patients with Angelman Syndrome (FANS): Study Protocol for a Randomized Controlled Trial
Research Square (Research Square)·2019· 1 citedOpen Access
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Frequent Co-authors

J. David Sweatt(20), Melinda Peters(8), Shenfeng Qiu(7), Joachim Herz(6), Justin H. Trotter(6), Umesh K. Jinwal(6), Chad A. Dickey(6), Justin Rogers(5), Uwe Beffert(5), Jessica L. Banko(5), Dawna L. Armstrong(4), G. William Rebeck(4), John Koren(4), John C. O’Leary(4), Kimberly Korwek(4), Joel C. Selcher(4), Laura J. Blair(4), Jill M. Christian(3), Bisong Xu(3), Richard Paylor(3)