Martin C. Carey

Trinity College Dublin — Ireland

Medicine · Health Sciences

69h-index17.0kcitations728works0.02yr avg
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Research Topics

Drug Transport and Resistance Mechanisms(102), Cholesterol and Lipid Metabolism(48), Gallbladder and Bile Duct Disorders(44), Liver Disease Diagnosis and Treatment(42), Pediatric Hepatobiliary Diseases and Treatments(39)

Publications728 total

Investigating the Toll-like Receptor 3 (TLR3) Leu412phe (TLR3 L412F) Single Nucleotide Polymorphism in the Pathogenesis of Long COVID
2024
Book review: Australian Indigenous studies: Research and practice, by Terry Moore, Carol Pybus, Mitchell Rolls and David Moltow 2016, Peter Lang AG, Bern, Switzerland, 286pp, ISBN 9783034322454 (pbk)
Murdoch Research Repository (Murdoch University)·2017
Influence of Phosphatidylcholine and Calcium on Self-Association and Bile Salt Mixed Micellar Binding of the Natural Bile Pigment, Bilirubin Ditaurate
Biochemistry·2015· 5 cited
Self-Assembly of Aqueous Bilirubin Ditaurate, a Natural Conjugated Bile Pigment, to Contraposing Enantiomeric Dimers and M(−) and P(+) Tetramers and Their Selective Hydrophilic Disaggregation by Monomers and Micelles of Bile Salts
Biochemistry·2015· 4 cited
Impaired cholecystokinin-induced gallbladder emptying incriminated in spontaneous “black” pigment gallstone formation in germfree Swiss Webster mice
American Journal of Physiology-Gastrointestinal and Liver Physiology·2014· 15 citedOpen Access
Impaired cholecystokinin-induced gallbladder emptying incriminated in spontaneous “black” pigment gallstone formation in germfree Swiss Webster mice
DSpace@MIT (Massachusetts Institute of Technology)·2014Open Access
Metastable and equilibrium phase diagrams of unconjugated bilirubin IXα as functions of pH in model bile systems: Implications for pigment gallstone formation
American Journal of Physiology-Gastrointestinal and Liver Physiology·2014· 12 citedOpen Access
Tu1955 Impaired Cholecystokinin-Induced Gallbladder Emptying Implicated in Spontaneous “Black” Pigment Gallstone Formation in Germfree Swiss Webster Mice
Gastroenterology·2014
An Analysis of the Role of the Indigenous Microbiota in Cholesterol Gallstone Pathogenesis
PLoS ONE·2013· 51 citedOpen Access
Roles of cholesterol and bile salts in the pathogenesis of gallbladder hypomotility and inflammation: cholecystitis is not caused by cystic duct obstruction
Neurogastroenterology & Motility·2013· 28 cited
Disruption of gallbladder smooth muscle function is an early feature in the development of cholesterol gallstone disease
Neurogastroenterology & Motility·2012· 81 citedOpen Access
Role of parietal (gallbladder musocal) factors in the formation of ‘black’ pigment gallstones. A response to A. Cariati and E. Piromalli's letter to the editor
Clinics and Research in Hepatology and Gastroenterology·2012· 1 cited
Augmented cholesterol absorption and sarcolemmal sterol enrichment slow small intestinal transit in mice, contributing to cholesterol cholelithogenesis
The Journal of Physiology·2012· 19 citedOpen Access
New pathophysiological concepts underlying pathogenesis of pigment gallstones
Clinics and Research in Hepatology and Gastroenterology·2011· 133 citedOpen Access
Hyperhomocysteinemia from trimethylation of hepatic phosphatidylethanolamine during cholesterol cholelithogenesis in inbred mice
Hepatology·2011· 8 citedOpen Access
Striking Alterations in Bilirubin and Bile Salt Solution Chemistry of Gallbladder Bile in a Germ-Free Mouse Model of “Black” Pigment Gallstones
Gastroenterology·2011· 1 citedOpen Access
Pathophysiological preconditions promoting mixed “black” pigment plus cholesterol gallstones in a ΔF508 mouse model of cystic fibrosis
American Journal of Physiology-Gastrointestinal and Liver Physiology·2010· 19 cited
485 Il10 Deficiency Markedly Ameliorates and Provides Prolonged Protection Against Cholesterol Gallstone Formation in a Susceptible Mouse Model
Gastroenterology·2010Open Access
S1777 Toll-Like Receptor 2 (TLR2) Deficiency Markedly Diminishes Gallstone Formation in C57BL/6 Mice Fed a Lithogenic Diet
Gastroenterology·2009
Cri-du-Chat Syndrome (Chromosome 5 Short Arm Deletion)
2009
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Frequent Co-authors

Norman A. Mazer(9), David Q.‐H. Wang(8), George B. Benedek(7), Donald Small(5), Beverly Paigen(5), David E. Cohen(4), Paul J. Missel(4), Frank Lammert(4), Olle Hernell(2), Joan E. Staggers(2), Sudha B. Biddinger(2), Joel T. Haas(2), C. Ronald Kahn(2), Charles Y. Young(2), Sum P. Lee(2), D Q Wang(2), Alan C. Moses(2), Jeffrey S. Flier(2), Kirk J. Maurer(2), James G. Fox(2)