Scott A. Armstrong

Boston Children's Hospital — United States

Medicine · Health Sciences

92h-index40.1kcitations654works2.72yr avg
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Research Topics

Acute Myeloid Leukemia Research(286), Protein Degradation and Inhibitors(145), Epigenetics and DNA Methylation(107), Cancer-related gene regulation(60), Acute Lymphoblastic Leukemia research(59)

Publications654 total

Supplementary Figures S1-S12 from Mutant NPM1 Directly Regulates Oncogenic Transcription in Acute Myeloid Leukemia
2025Open Access
Supplementary Tables S1-S14 from Mutant NPM1 Directly Regulates Oncogenic Transcription in Acute Myeloid Leukemia
2025Open Access
Supplementary Table 4 from A Molecular Switch between Mammalian MLL Complexes Dictates Response to Menin–MLL Inhibition
2025Open Access
Supplementary Table 1 from A Molecular Switch between Mammalian MLL Complexes Dictates Response to Menin–MLL Inhibition
2025Open Access
Supplementary Data from MOZ and Menin–MLL Complexes Are Complementary Regulators of Chromatin Association and Transcriptional Output in Gastrointestinal Stromal Tumor
2025Open Access
Supplementary Table 3 from A Molecular Switch between Mammalian MLL Complexes Dictates Response to Menin–MLL Inhibition
2025Open Access
Supplementary Figures from A Molecular Switch between Mammalian MLL Complexes Dictates Response to Menin–MLL Inhibition
2025Open Access
Supplementary Data from MOZ and Menin–MLL Complexes Are Complementary Regulators of Chromatin Association and Transcriptional Output in Gastrointestinal Stromal Tumor
2025Open Access
Supplementary Table 2 from A Molecular Switch between Mammalian MLL Complexes Dictates Response to Menin–MLL Inhibition
2025Open Access
Supplementary Data from MOZ and Menin–MLL Complexes Are Complementary Regulators of Chromatin Association and Transcriptional Output in Gastrointestinal Stromal Tumor
2025Open Access
Supplementary Data from MOZ and Menin–MLL Complexes Are Complementary Regulators of Chromatin Association and Transcriptional Output in Gastrointestinal Stromal Tumor
2025Open Access
Data from MOZ and Menin–MLL Complexes Are Complementary Regulators of Chromatin Association and Transcriptional Output in Gastrointestinal Stromal Tumor
2025Open Access
Supplementary Data from MOZ and Menin–MLL Complexes Are Complementary Regulators of Chromatin Association and Transcriptional Output in Gastrointestinal Stromal Tumor
2025Open Access
Targeting BRD9 in KMT2A-rearranged Acute Myeloid Leukemia to circumvent MEN1 inhibitor resistance
Blood·2025Open Access
The KMT2A-PTD oncoprotein depends on ENL but not menin to drive AML gene expression
Blood·2025
Supplementary Figure S3 from KAT6A and KAT7 Histone Acetyltransferase Complexes Are Molecular Dependencies and Therapeutic Targets in <i>NUP98</i>-Rearranged Acute Myeloid Leukemia
2025Open Access
Supplementary Figures S1-8 from Catalytic Inhibition of KAT6/KAT7 Enhances the Efficacy and Overcomes Primary and Acquired Resistance to Menin Inhibitors in MLL Leukemia
2025Open Access
Supplementary Figure S4 from KAT6A and KAT7 Histone Acetyltransferase Complexes Are Molecular Dependencies and Therapeutic Targets in <i>NUP98</i>-Rearranged Acute Myeloid Leukemia
2025Open Access
Supplementary Methods and Results from KAT6A and KAT7 Histone Acetyltransferase Complexes Are Molecular Dependencies and Therapeutic Targets in <i>NUP98</i>-Rearranged Acute Myeloid Leukemia
2025Open Access
Supplementary Tables S1-14 from Catalytic Inhibition of KAT6/KAT7 Enhances the Efficacy and Overcomes Primary and Acquired Resistance to Menin Inhibitors in MLL Leukemia
2025Open Access
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Frequent Co-authors

Andrei V. Krivtsov(16), Amit Sinha(8), Andrew L. Kung(7), Todd R. Golub(6), Stephen E. Sallan(6), Ross L. Levine(6), Lewis B. Silverman(5), Stanley J. Korsmeyer(5), Lars Bullinger(5), Richard P. Koche(5), Omar Abdel‐Wahab(5), Zhaohui Feng(4), Joerg Faber(4), William C. Hahn(4), Nan Zhu(4), Gerard M. McGeehan(4), Chun‐Wei Chen(4), James E. Bradner(4), Guo Wei(3), Rob Pieters(3)