Laura Riva

Wellcome Sanger Institute — United Kingdom

Biochemistry, Genetics and Molecular Biology · Life Sciences

21h-index2.4kcitations86works0.82yr avg
OpenAlex↗ORCID↗Google Scholar↗Lab page↗

Accepting Students?

No reports yet. Know if this professor is taking students?

Research Topics

Cancer Genomics and Diagnostics(38), Cancer Cells and Metastasis(13), Epigenetics and DNA Methylation(9), Bioinformatics and Genomic Networks(8), Genetic factors in colorectal cancer(8)

Publications86 total

Environmental carcinogens often exacerbate endogenous mutagenic processes to enhance tumor promotion
Cell Reports·2025· 2 citedOpen Access
Mediastinal staging of nonsmall cell lung cancer: what's new?
Breathe·2025· 4 citedOpen Access
Data from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 2 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 6 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Table 2 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 5 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 7 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 3 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 1 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Dataset from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 4 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 8 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Table 1 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 9 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
Cancer Discovery·2025· 6 citedOpen Access
Highlights from the 1st European cancer dependency map symposium and workshop
FEBS Letters·2023· 11 citedOpen Access
Supplementary Methods, Figure Legends from <i>In Vivo</i> Genetic Screens of Patient-Derived Tumors Revealed Unexpected Frailty of the Transformed Phenotype
2023Open Access
Data from <i>In Vivo</i> Genetic Screens of Patient-Derived Tumors Revealed Unexpected Frailty of the Transformed Phenotype
2023Open Access
Data from <i>In Vivo</i> Genetic Screens of Patient-Derived Tumors Revealed Unexpected Frailty of the Transformed Phenotype
2023Open Access
page 1 of 5Next →

Frequent Co-authors

Pier Giuseppe Pelicci(19), Giorgio Melloni(12), David J. Adams(9), Lucilla Luzi(9), Leslie A. Lange(5), Ludmil B. Alexandrov(5), Gaetano Ivan Dellino(5), Luca Mazzarella(5), Margherita Bodini(5), Chiara Ronchini(5), Ilaria Iacobucci(5), Giovanni Martinelli(5), David C. Wedge(4), Ernest Fraenkel(4), Luisa Lanfrancone(4), Giuseppe Curigliano(4), Anna Russo(4), Sara Volorio(4), Serena Lavorgna(4), Francesco Lo‐Coco(4)