Joseph Tuinei

University of Utah — United States

Medicine · Health Sciences

12h-index1.5kcitations20works0.02yr avg
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Research Topics

Adipose Tissue and Metabolism(10), Cardiovascular Function and Risk Factors(9), Metabolism, Diabetes, and Cancer(7), Mitochondrial Function and Pathology(4), Pancreatic function and diabetes(4)

Publications20 total

Maintaining Myocardial Glucose Utilization in Diabetic Cardiomyopathy Accelerates Mitochondrial Dysfunction
Diabetes·2020· 67 citedOpen Access
Maintaining Myocardial Glucose Utilization in Diabetic Cardiomyopathy Accelerates Mitochondrial Dysfunction
2020· 4 citedOpen Access
Maintaining Myocardial Glucose Utilization in Diabetic Cardiomyopathy Accelerates Mitochondrial Dysfunction
2020Open Access
Glucose transporter 4-deficient hearts develop maladaptive hypertrophy in response to physiological or pathological stresses
American Journal of Physiology-Heart and Circulatory Physiology·2017· 59 citedOpen Access
Antioxidant treatment normalizes mitochondrial energetics and myocardial insulin sensitivity independently of changes in systemic metabolic homeostasis in a mouse model of the metabolic syndrome
Journal of Molecular and Cellular Cardiology·2015· 35 cited
Enhanced Cardiac Akt/Protein Kinase B Signaling Contributes to Pathological Cardiac Hypertrophy in Part by Impairing Mitochondrial Function via Transcriptional Repression of Mitochondrion-Targeted Nuclear Genes
Molecular and Cellular Biology·2014· 89 citedOpen Access
UCP3 Regulates Cardiac Efficiency and Mitochondrial Coupling in High Fat–Fed Mice but Not in Leptin-Deficient Mice
Diabetes·2012· 62 citedOpen Access
Genetic loss of insulin receptors worsens cardiac efficiency in diabetes
Journal of Molecular and Cellular Cardiology·2012· 65 citedOpen Access
The Intermountain Risk Score Stratifies Mortality Risk in Heart Failure Patients Receiving an ICD Based on Pre-Implant Laboratory Values
Journal of Cardiac Failure·2011· 2 cited
Central Leptin Signaling Is Required to Normalize Myocardial Fatty Acid Oxidation Rates in Caloric-Restricted <i>ob/ob</i> Mice
Diabetes·2011· 82 citedOpen Access
PPARγ-induced cardiolipotoxicity in mice is ameliorated by PPARα deficiency despite increases in fatty acid oxidation
Journal of Clinical Investigation·2010· 2 citedOpen Access
Abstract 17402: Hypothalamic Leptin Resistance Contributes to Increased Myocardial Fatty Acid Oxidation Rates in Obesity
Circulation·2010
PPARγ-induced cardiolipotoxicity in mice is ameliorated by PPARα deficiency despite increases in fatty acid oxidation
Journal of Clinical Investigation·2010· 155 citedOpen Access
An APPL1-AMPK signaling axis mediates beneficial metabolic effects of adiponectin in the heart
American Journal of Physiology-Endocrinology and Metabolism·2010· 117 citedOpen Access
Abstract 5379: Increased Myocardial Fatty Acid Uptake by Acyl-CoA Synthetase 1 Impairs Mitochondrial Function and Dynamics
Circulation·2008
Type 1 Diabetic Akita Mouse Hearts Are Insulin Sensitive but Manifest Structurally Abnormal Mitochondria That Remain Coupled Despite Increased Uncoupling Protein 3
Diabetes·2008· 177 citedOpen Access
Ceramide is a cardiotoxin in lipotoxic cardiomyopathy
Journal of Lipid Research·2008· 401 citedOpen Access
Abstract 647: Uncoupling Protein 3 Does Not Regulate Myocardial Fatty Acid Oxidation Rates But Regulates ROS Production and Cardiac Efficiency
Circulation·2007
Abstract 509: Myocardial Fatty Acid Oxidation Rates Remain Elevated in ob/ob Mice Despite Reversal of Obesity and Diabetes by Caloric Restriction
Circulation·2007· 2 cited
A Conserved Role for Phosphatidylinositol 3-Kinase but Not Akt Signaling in Mitochondrial Adaptations that Accompany Physiological Cardiac Hypertrophy
Cell Metabolism·2007· 137 citedOpen Access

Frequent Co-authors

E. Dale Abel(17), Sheldon E. Litwin(8), Adam R. Wende(7), Heiko Bugger(6), Sihem Boudina(5), Heather Theobald(5), Benjamin Wayment(5), Curtis D. Olsen(5), John C. Schell(4), Crystal Sloan(4), Renata O. Pereira(4), Mark E. Pepin(4), Manoja K. Brahma(4), Jonathan Buchanan(3), Ira J. Goldberg(3), Brian T. O’Neill(3), Li Wang(3), Jamie Soto(3), Chae‐Myeong Ha(3), Oleh Khalimonchuk(3)