B. Mark Woerner

Pediatrics and Genetics — United States

Biochemistry, Genetics and Molecular Biology · Life Sciences

20h-index5.7kcitations73works0.02yr avg
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Research Topics

Cancer, Hypoxia, and Metabolism(16), Metabolism, Diabetes, and Cancer(14), Pancreatic function and diabetes(13), Chemokine receptors and signaling(13), Cancer Mechanisms and Therapy(13)

Publications73 total

Data from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Data from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Table 2 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Table 1 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Figure 1 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Table 2 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Table 3 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Table 4 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Table 4 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Figure 3 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Figure 2 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Figure Legend from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Table 3 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Figure 3 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Figure 2 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Table 1 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Figure 1 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Figure Legend from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Figures S1-S3 from Targeted Inhibition of Cyclic AMP Phosphodiesterase-4 Promotes Brain Tumor Regression
2023Open Access
Data from Targeted Inhibition of Cyclic AMP Phosphodiesterase-4 Promotes Brain Tumor Regression
2023Open Access
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Frequent Co-authors

Joshua B. Rubin(35), David Piwnica‐Worms(21), Erin Jackson(18), Arie Perry(16), Jingqin Luo(16), Alane Koki(12), Kristin R. Brown(12), Sonika Dahiya(12), Paul S. Mischel(12), Jeffrey Benovic(12), Jaime L. Masferrer(11), Nicole M. Warrington(11), Karen Seibert(5), Robert A. Soslow(5), Andrew J. Dannenberg(5), Rajarshi Sengupta(5), Dorothy Edwards(4), Andrew L. Kung(4), Girish Daginakatte(4), Biplab Dasgupta(4)