Erin Jackson

Dalhousie University — Canada

Biochemistry, Genetics and Molecular Biology · Life Sciences

27h-index2.2kcitations94works2.02yr avg
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Research Topics

Cancer, Hypoxia, and Metabolism(16), Metabolism, Diabetes, and Cancer(14), Pancreatic function and diabetes(13), Cancer Mechanisms and Therapy(8), Hormonal Regulation and Hypertension(7)

Publications94 total

Nickel‐Catalyzed <i>O</i>‐Arylation of <i>N</i>‐Protected Amino Alcohols with (Hetero)aryl Chlorides
Chemistry - A European Journal·2024· 3 citedOpen Access
Comparison of two‐dimensional imaging to three‐dimensional modeling of intrahepatic portosystemic shunts using computed tomography angiography
Veterinary Radiology & Ultrasound·2024· 1 citedOpen Access
The soluble guanylyl cyclase pathway is inhibited to evade androgen deprivation-induced senescence and enable progression to castration resistance
2023· 3 citedOpen Access
Data from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Data from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Table 2 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Table 1 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Figure 1 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Table 2 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Table 3 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Table 4 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Table 4 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Figure 3 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Figure 2 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Figure Legend from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Table 3 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Figure 3 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Figure 2 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Table 1 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
Supplementary Figure 1 from Suppression of G-protein–Coupled Receptor Kinase 3 Expression Is a Feature of Classical GBM That Is Required for Maximal Growth
2023Open Access
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Frequent Co-authors

David Piwnica‐Worms(10), Glen E. Mott(9), Joshua B. Rubin(6), Katie E. Cherry(6), Karri S. Hawley(5), Russell E. Johnson(4), Douglas S. Lewis(4), Jeong-Sun Ju(3), Sara Miller(3), Conrad C. Weihl(3), G M Fless(3), D Pfaffinger(3), Jeffrey R. Leonard(3), Henry C. McGill(3), C. Alex McMahan(3), Jayne Marasa(2), Shyam Rao(2), Rajarshi Sengupta(2), B. Mark Woerner(2), Tao Sun(2)